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Human blood platelet adhesion to artery subendothelium is mediated by factor VIII-Von Willebrand factor bound to the subendothelium.

作者信息

Sakariassen K S, Bolhuis P A, Sixma J J

出版信息

Nature. 1979 Jun 14;279(5714):636-8. doi: 10.1038/279636a0.

DOI:10.1038/279636a0
PMID:313016
Abstract
摘要

相似文献

1
Human blood platelet adhesion to artery subendothelium is mediated by factor VIII-Von Willebrand factor bound to the subendothelium.人血小板对动脉内皮下层的黏附是由结合于内皮下层的凝血因子VIII-血管性血友病因子介导的。
Nature. 1979 Jun 14;279(5714):636-8. doi: 10.1038/279636a0.
2
Adhesion of blood platelets to human arterial subendothelium: role of factor VIII-Von Willebrand factor.血小板与人动脉内皮下层的黏附:因子VIII-血管性血友病因子的作用。
Haemostasis. 1979;8(3-5):312-23. doi: 10.1159/000214321.
3
Culture-produced subendothelium. II. Effect of plasma, F.VIIIR:WF and fibronectin on interaction of normal platelets with normal and von Willebrand porcine aortic subendothelium.培养产生的内皮下层。II. 血浆、F.VIIIR:WF和纤连蛋白对正常血小板与正常及血管性血友病猪主动脉内皮下层相互作用的影响。
Thromb Res. 1982 Nov 1;28(3):299-311. doi: 10.1016/0049-3848(82)90113-x.
4
Lack of thromboxane A2 synthesis in platelet aggregation induced by factor VIII/von Willebrand factor.在因子VIII/血管性血友病因子诱导的血小板聚集中血栓素A2合成的缺乏
Thromb Res. 1981 Nov 1;24(3):245-51. doi: 10.1016/0049-3848(81)90094-3.
5
Binding of factor VIII-von Willebrand factor to human arterial subendothelium precedes increased platelet adhesion and enhances platelet spreading.凝血因子VIII-血管性血友病因子与人类动脉内皮下层的结合先于血小板黏附增加,并增强血小板铺展。
J Lab Clin Med. 1981 Apr;97(4):568-76.
6
Factor VIII/von Willebrand factor in subendothelium mediates platelet adhesion.内皮下的凝血因子VIII/血管性血友病因子介导血小板黏附。
Blood. 1985 Apr;65(4):823-31.
7
Support of ristocetin-induced platelet aggregation by procoagulant-inactive and plasmin-cleaved forms of human factor VIII/von Willebrand factor.人凝血因子VIII/血管性血友病因子的促凝无活性形式和纤溶酶裂解形式对瑞斯托菌素诱导的血小板聚集的支持作用。
Blood. 1980 Jan;55(1):101-8.
8
Bernard-Soulier syndrome: a new platelet glycoprotein abnormality. Its relationship with platelet adhesion to subendothelium and with the factor VIII von Willebrand protein.伯纳德-索利尔综合征:一种新的血小板糖蛋白异常。它与血小板黏附于内皮下以及与因子VIII血管性血友病蛋白的关系。
J Lab Clin Med. 1976 Apr;87(4):586-96.
9
Role of von Willebrand factor in platelet adhesion to the subendothelium.血管性血友病因子在血小板黏附于内皮下的作用。
Br J Haematol. 1983 May;54(1):1-9. doi: 10.1111/j.1365-2141.1983.tb02061.x.
10
von Willebrand factor contained in factor VIII concentrates of different purities supports platelet adhesion in blood samples from a heterogeneous group of patients with von Willebrand disease.不同纯度的凝血因子 VIII 浓缩物中所含的血管性血友病因子支持来自异质性血管性血友病患者组的血样中的血小板黏附。
Haematologica. 1998 Nov;83(11):1009-14.

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Transamniotic Delivery of Coagulation Factor VIII mRNA: A Step Toward a Potential Novel Strategy for the Perinatal Management of Hemophilia A.凝血因子VIII mRNA的经羊膜递送:迈向血友病A围产期管理潜在新策略的一步。
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2
Low Bone Mineral Density in Hemophiliacs.血友病患者的低骨矿物质密度
Front Med (Lausanne). 2022 Feb 2;9:794456. doi: 10.3389/fmed.2022.794456. eCollection 2022.
3
Type IV Pili of Streptococcus sanguinis Contribute to Pathogenesis in Experimental Infective Endocarditis.
血链球菌 IV 型菌毛有助于实验性感染性心内膜炎的发病机制。
Microbiol Spectr. 2021 Dec 22;9(3):e0175221. doi: 10.1128/Spectrum.01752-21. Epub 2021 Nov 10.
4
Simulation of thrombosis in a stenotic microchannel: The effects of vWF-enhanced shear activation of platelets.狭窄微通道内血栓形成的模拟:血管性血友病因子增强血小板剪切激活的影响。
Int J Eng Sci. 2020 Feb;147. doi: 10.1016/j.ijengsci.2019.103206. Epub 2019 Dec 20.
5
The Role of Anesthetic Selection in Perioperative Bleeding.麻醉选择在围手术期出血中的作用。
Biomed Res Int. 2021 May 7;2021:5510634. doi: 10.1155/2021/5510634. eCollection 2021.
6
Endothelial cells and coagulation.内皮细胞与凝血。
Cell Tissue Res. 2022 Mar;387(3):391-398. doi: 10.1007/s00441-021-03471-2. Epub 2021 May 20.
7
The Effect of a Polyester Nanofibrous Membrane with a Fibrin-Platelet Lysate Coating on Keratinocytes and Endothelial Cells in a Co-Culture System.在共培养系统中,具有纤维蛋白 - 血小板裂解物涂层的聚酯纳米纤维膜对角质形成细胞和内皮细胞的影响。
Nanomaterials (Basel). 2021 Feb 11;11(2):457. doi: 10.3390/nano11020457.
8
Development of a dual hybrid AAV vector for endothelial-targeted expression of von Willebrand factor.开发一种双杂交 AAV 载体,用于内皮细胞靶向表达血管性血友病因子。
Gene Ther. 2023 Apr;30(3-4):245-254. doi: 10.1038/s41434-020-00218-6. Epub 2021 Jan 17.
9
von Willebrand factor promotes platelet-induced metastasis of osteosarcoma through activation of the VWF-GPIb axis.血管性血友病因子通过激活VWF-GPIb轴促进血小板诱导的骨肉瘤转移。
J Bone Oncol. 2020 Oct 8;25:100325. doi: 10.1016/j.jbo.2020.100325. eCollection 2020 Dec.
10
Loss of GPVI and GPIbα contributes to trauma-induced platelet dysfunction in severely injured patients.GPVI 和 GPIbα 的缺失导致严重创伤患者的血小板功能障碍。
Blood Adv. 2020 Jun 23;4(12):2623-2630. doi: 10.1182/bloodadvances.2020001776.