Enigma of Inflammasome Activation by Kinases.
作者信息
Shen Ying H, Abe Jun-Ichi
机构信息
From the Michael E. Debakey Department of Surgery, Baylor College of Medicine, Houston, TX (Y.H.S.).
Department of Cardiology, University of Texas MD Anderson Cancer Center, Houston, TX (J.-i.A.).
出版信息
Arterioscler Thromb Vasc Biol. 2019 Aug;39(8):1501-1503. doi: 10.1161/ATVBAHA.119.312965. Epub 2019 Jul 24.
Abstract
摘要
相似文献
1
Enigma of Inflammasome Activation by Kinases.激酶激活炎性小体之谜
Arterioscler Thromb Vasc Biol. 2019 Aug;39(8):1501-1503. doi: 10.1161/ATVBAHA.119.312965. Epub 2019 Jul 24.
2
MARK4 regulates NLRP3 positioning and inflammasome activation through a microtubule-dependent mechanism.MARK4 通过微管依赖性机制调节 NLRP3 的定位和炎症小体的激活。
Nat Commun. 2017 Jun 28;8:15986. doi: 10.1038/ncomms15986.
3
Regulation and Function of the Nucleotide Binding Domain Leucine-Rich Repeat-Containing Receptor, Pyrin Domain-Containing-3 Inflammasome in Lung Disease.含核苷酸结合结构域富含亮氨酸重复序列受体、含吡啉结构域的3炎性小体在肺部疾病中的调控与功能
Am J Respir Cell Mol Biol. 2016 Feb;54(2):151-60. doi: 10.1165/rcmb.2015-0231TR.
4
Inflammasome Signaling and Other Factors Implicated in Atherosclerosis Development and Progression.炎症小体信号转导与其他与动脉粥样硬化发生发展相关的因素。
Curr Pharm Des. 2020;26(22):2583-2590. doi: 10.2174/1381612826666200504115045.
5
C1q/TNF-related protein-9 attenuates atherosclerosis through AMPK-NLRP3 inflammasome singling pathway.C1q/TNF 相关蛋白-9 通过 AMPK-NLRP3 炎性小体信号通路减轻动脉粥样硬化。
Int Immunopharmacol. 2019 Dec;77:105934. doi: 10.1016/j.intimp.2019.105934. Epub 2019 Nov 11.
6
Activation of NLRP3 Inflammasome Promotes Foam Cell Formation in Vascular Smooth Muscle Cells and Atherogenesis Via HMGB1.NLRP3 炎性小体的激活通过 HMGB1 促进血管平滑肌细胞泡沫细胞形成和动脉粥样硬化。
J Am Heart Assoc. 2018 Oct 2;7(19):e008596. doi: 10.1161/JAHA.118.008596.
7
The RIP1-RIP3 complex initiates mitochondrial fission to fuel NLRP3.RIP1-RIP3 复合物启动线粒体裂变以供应 NLRP3。
Nat Immunol. 2014 Dec;15(12):1100-2. doi: 10.1038/ni.3030.
8
Mechanism and Regulation of NLRP3 Inflammasome Activation.NLRP3炎性小体激活的机制与调控
Trends Biochem Sci. 2016 Dec;41(12):1012-1021. doi: 10.1016/j.tibs.2016.09.002. Epub 2016 Sep 23.
9
Targeting NLRP3 (Nucleotide-Binding Domain, Leucine-Rich-Containing Family, Pyrin Domain-Containing-3) Inflammasome in Cardiovascular Disorders.靶向 NLRP3（核苷酸结合域、富含亮氨酸重复序列、含pyrin 结构域蛋白 3）炎症小体治疗心血管疾病。
Arterioscler Thromb Vasc Biol. 2018 Dec;38(12):2765-2779. doi: 10.1161/ATVBAHA.118.311916.
10
Inhibitory effects of colchicine on inflammasomes.秋水仙碱对炎性小体的抑制作用。
Atherosclerosis. 2018 Jun;273:153-154. doi: 10.1016/j.atherosclerosis.2018.02.026. Epub 2018 Mar 2.
引用本文的文献
1
High-Density Lipoproteins at the Interface between the NLRP3 Inflammasome and Myocardial Infarction.NLRP3炎性小体与心肌梗死之间界面处的高密度脂蛋白
Int J Mol Sci. 2024 Jan 20;25(2):1290. doi: 10.3390/ijms25021290.
2
A detailed molecular network map and model of the NLRP3 inflammasome.NLRP3 炎症小体的详细分子网络图谱和模型。
Front Immunol. 2023 Nov 15;14:1233680. doi: 10.3389/fimmu.2023.1233680. eCollection 2023.
3
Changes in plasma IRAK-M in patients with prediabetes and its relationship with related metabolic indexes: a cross-sectional study.空腹血糖受损患者血浆 IRAK-M 的变化及其与相关代谢指标的关系：一项横断面研究。
J Int Med Res. 2022 Aug;50(8):3000605221111275. doi: 10.1177/03000605221111275.
本文引用的文献
1
Effects of phosphorylation on the NLRP3 inflammasome.磷酸化对 NLRP3 炎性小体的影响。
Arch Biochem Biophys. 2019 Jul 30;670:43-57. doi: 10.1016/j.abb.2019.02.020. Epub 2019 Mar 5.
2
Podocyte NADPH Oxidase 5 Promotes Renal Inflammation Regulated by the Toll-Like Receptor Pathway.足细胞 NADPH 氧化酶 5 通过 Toll 样受体通路促进肾脏炎症。
Antioxid Redox Signal. 2019 May 20;30(15):1817-1830. doi: 10.1089/ars.2017.7402. Epub 2018 Sep 8.
3
NLRP3 Phosphorylation Is an Essential Priming Event for Inflammasome Activation.NLRP3 磷酸化是炎症小体激活的必要初始事件。
Mol Cell. 2017 Oct 5;68(1):185-197.e6. doi: 10.1016/j.molcel.2017.08.017. Epub 2017 Sep 21.
4
CLICs-dependent chloride efflux is an essential and proximal upstream event for NLRP3 inflammasome activation.氯离子细胞内通道蛋白（CLICs）依赖性氯离子外流是NLRP3炎性小体激活的一个重要且直接的上游事件。
Nat Commun. 2017 Aug 4;8(1):202. doi: 10.1038/s41467-017-00227-x.
5
Protein kinase D at the Golgi controls NLRP3 inflammasome activation.高尔基体中的蛋白激酶D控制NLRP3炎性小体的激活。
J Exp Med. 2017 Sep 4;214(9):2671-2693. doi: 10.1084/jem.20162040. Epub 2017 Jul 17.
6
MARK4 regulates NLRP3 positioning and inflammasome activation through a microtubule-dependent mechanism.MARK4 通过微管依赖性机制调节 NLRP3 的定位和炎症小体的激活。
Nat Commun. 2017 Jun 28;8:15986. doi: 10.1038/ncomms15986.
7
The NLRP3 inflammasome and bruton's tyrosine kinase in platelets co-regulate platelet activation, aggregation, and in vitro thrombus formation.血小板中的NLRP3炎性小体和布鲁顿酪氨酸激酶共同调节血小板活化、聚集及体外血栓形成。
Biochem Biophys Res Commun. 2017 Jan 29;483(1):230-236. doi: 10.1016/j.bbrc.2016.12.161. Epub 2016 Dec 26.
8
Pyk2 activates the NLRP3 inflammasome by directly phosphorylating ASC and contributes to inflammasome-dependent peritonitis.Pyk2通过直接磷酸化ASC激活NLRP3炎性小体，并导致炎性小体依赖性腹膜炎。
Sci Rep. 2016 Oct 31;6:36214. doi: 10.1038/srep36214.
9
NLRP3 inflammasome inhibition is disrupted in a group of auto-inflammatory disease CAPS mutations.NLRP3 炎性体抑制在一组自身炎症性疾病 CAPS 突变中被破坏。
Nat Immunol. 2016 Oct;17(10):1176-86. doi: 10.1038/ni.3538. Epub 2016 Aug 22.
10
Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores.炎性小体激活的gasdermin D通过形成膜孔导致细胞焦亡。
Nature. 2016 Jul 7;535(7610):153-8. doi: 10.1038/nature18629.
Zotero 插件