自身免疫小鼠的T细胞受体：C3H-gld/gld小鼠中新型T细胞亚群的功能与分子分析 - Suppr

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超能文献

T cell receptors of autoimmune mice: functional and molecular analysis of novel T cell subsets in C3H-gld/gld mice.

作者信息

Yui K, Hashimoto Y, Greene M I

机构信息

Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, Philadelphia.

出版信息

Immunol Res. 1988;7(2):173-88. doi: 10.1007/BF02918099.

DOI:10.1007/BF02918099

PMID:3134499

Abstract

摘要

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T cell receptors of autoimmune mice: functional and molecular analysis of novel T cell subsets in C3H-gld/gld mice.自身免疫小鼠的T细胞受体：C3H-gld/gld小鼠中新型T细胞亚群的功能与分子分析

Immunol Res. 1988;7(2):173-88. doi: 10.1007/BF02918099.

The expanded populations of CD4-CD8- T cell receptor alpha/beta+ T cells associated with the lpr and gld mutations are CD2-.与lpr和gld突变相关的CD4-CD8-T细胞受体α/β+T细胞扩增群体为CD2阴性。

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Surface receptors involved in human T lymphocyte activation.参与人类T淋巴细胞活化的表面受体。

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Ectopic expression of B7-1 (CD80) on T lymphocytes in autoimmune lpr and gld mice.自身免疫性lpr和gld小鼠T淋巴细胞上B7-1（CD80）的异位表达。

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Lymphocyte subsets and surface molecules in man.人类淋巴细胞亚群及表面分子

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Effect of xid on autoimmune C3H-gld/gld mice.Xid对自身免疫性C3H-gld/gld小鼠的影响。

Cell Immunol. 1987 Jun;107(1):249-55. doi: 10.1016/0008-8749(87)90284-x.

A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice.一种新的突变体gld，它在C3H/HeJ小鼠中引发淋巴细胞增殖和自身免疫。

J Exp Med. 1984 Jan 1;159(1):1-20. doi: 10.1084/jem.159.1.1.

Defective T cells from gld mice play a pivotal role in development of Thy-1.2+B220+ cells and autoimmunity.来自gld小鼠的缺陷性T细胞在Thy-1.2+B220+细胞的发育和自身免疫中起关键作用。

J Immunol. 1994 Dec 15;153(12):5855-64.

Biology of the T cell.T细胞生物学

Prog Clin Biol Res. 1986;224:11-20.

Functional distinctions between MRL-lpr and MRL-gld lymphocytes. Normal cells reverse the gld but not lpr immunoregulatory defect.MRL-lpr和MRL-gld淋巴细胞之间的功能差异。正常细胞可逆转gld而非lpr的免疫调节缺陷。

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引用本文的文献

Fas- and perforin-independent mechanism of cytotoxic T lymphocyte.细胞毒性T淋巴细胞的Fas和穿孔素非依赖机制

Immunol Res. 1998;17(1-2):89-93. doi: 10.1007/BF02786434.

Major histocompatibility complex class I-specific cytolytic T cells, derived from gld mice, lacking Thy-1, CD4, and CD8.源自gld小鼠的主要组织相容性复合体I类特异性细胞毒性T细胞，缺乏Thy-1、CD4和CD8。

Proc Natl Acad Sci U S A. 1989 Feb;86(3):1018-22. doi: 10.1073/pnas.86.3.1018.

CD4-CD8+ T-cell receptor-alpha beta+ major histocompatibility complex class-II-specific T-cell clones isolated from aged athymic mice.

Immunol Res. 1992;11(1):3-10. doi: 10.1007/BF02918603.

本文引用的文献

Transmembrane signalling by the T3-antigen receptor complex.

Immunol Today. 1985 Nov;6(11):328-31. doi: 10.1016/0167-5699(85)90129-X.

THE AKR THYMIC ANTIGEN AND ITS DISTRIBUTION IN LEUKEMIAS AND NERVOUS TISSUES.AKR胸腺抗原及其在白血病和神经组织中的分布。

J Exp Med. 1964 Sep 1;120(3):413-33. doi: 10.1084/jem.120.3.413.

Hybridoma cell lines secreting monoclonal antibodies to mouse H-2 and Ia antigens.分泌针对小鼠H-2和Ia抗原的单克隆抗体的杂交瘤细胞系。

J Immunol. 1980 Feb;124(2):533-40.

T-cell growth factor.T细胞生长因子。

Immunol Rev. 1980;51:337-57. doi: 10.1111/j.1600-065x.1980.tb00327.x.

Possible involvement of the OKT4 molecule in T cell recognition of class II HLA antigens. Evidence from studies of cytotoxic T lymphocytes specific for SB antigens.OKT4分子可能参与T细胞对II类HLA抗原的识别。来自针对SB抗原的细胞毒性T淋巴细胞研究的证据。

J Exp Med. 1982 Oct 1;156(4):1065-76. doi: 10.1084/jem.156.4.1065.

Significance of Lyt phenotypes: Lyt2 antibodies block activities of T cells that recognize class 1 major histocompatibility complex antigens regardless of their function.Lyt表型的意义：Lyt2抗体可阻断识别1类主要组织相容性复合体抗原的T细胞的活性，无论其功能如何。

Proc Natl Acad Sci U S A. 1981 Nov;78(11):7101-5. doi: 10.1073/pnas.78.11.7101.

T cell growth factor receptors. Quantitation, specificity, and biological relevance.T细胞生长因子受体。定量、特异性及生物学相关性。

J Exp Med. 1981 Nov 1;154(5):1455-74. doi: 10.1084/jem.154.5.1455.

B220: a B cell-specific member of th T200 glycoprotein family.B220：T200糖蛋白家族的一种B细胞特异性成员。

Nature. 1981 Feb 19;289(5799):681-3. doi: 10.1038/289681a0.

Abnormalities induced by the mutant gene Ipr: expansion of a unique lymphocyte subset.由突变基因Ipr诱导的异常：一种独特淋巴细胞亚群的扩增。

J Immunol. 1982 Dec;129(6):2612-5.

T-cell mitogens cause early changes in cytoplasmic free Ca2+ and membrane potential in lymphocytes.T细胞有丝分裂原可引起淋巴细胞胞质游离Ca2+和膜电位的早期变化。

Nature. 1982 Jan 7;295(5844):68-71. doi: 10.1038/295068a0.

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