The Colon as a Major Site of Immunoregulation by CD4 T Cell Subsets in the Steady State.
作者信息
Sher Alan, Kelsall Brian L
机构信息
Laboratory of Parasitic Diseases, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892; and.
Laboratory of Molecular Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892
出版信息
J Immunol. 2019 Oct 1;203(7):1683-1684. doi: 10.4049/jimmunol.1900960.
Abstract
摘要
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本文引用的文献
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Semin Immunol. 2013 Nov 30;25(5):352-7. doi: 10.1016/j.smim.2013.09.002. Epub 2013 Oct 31.
2
CD4(+) T-cell subsets in intestinal inflammation.肠道炎症中的 CD4(+) T 细胞亚群。
Immunol Rev. 2013 Mar;252(1):164-82. doi: 10.1111/imr.12039.
3
Induction of colitis by a CD4+ T cell clone specific for a bacterial epitope.由针对细菌表位的CD4 + T细胞克隆诱导结肠炎。
Proc Natl Acad Sci U S A. 2003 Dec 23;100(26):15830-5. doi: 10.1073/pnas.2534546100. Epub 2003 Dec 12.
4
Monoassociation of SCID mice with Helicobacter muridarum, but not four other enterics, provokes IBD upon receipt of T cells.将重度联合免疫缺陷（SCID）小鼠仅与鼠幽门螺杆菌而非其他四种肠道细菌进行单菌定植，在输入T细胞后会引发炎症性肠病（IBD）。
Gastroenterology. 2002 May;122(5):1346-54. doi: 10.1053/gast.2002.32959.
5
Cytotoxic T lymphocyte-associated antigen 4 plays an essential role in the function of CD25(+)CD4(+) regulatory cells that control intestinal inflammation.细胞毒性T淋巴细胞相关抗原4在控制肠道炎症的CD25(+)CD4(+)调节性细胞的功能中发挥着重要作用。
J Exp Med. 2000 Jul 17;192(2):295-302. doi: 10.1084/jem.192.2.295.
6
A critical role for transforming growth factor-beta but not interleukin 4 in the suppression of T helper type 1-mediated colitis by CD45RB(low) CD4+ T cells.转化生长因子-β而非白细胞介素4在CD45RB(low) CD4+ T细胞抑制1型辅助性T细胞介导的结肠炎中起关键作用。
J Exp Med. 1996 Jun 1;183(6):2669-74. doi: 10.1084/jem.183.6.2669.
7
Ulcerative colitis-like disease in mice with a disrupted interleukin-2 gene.白细胞介素-2基因缺失小鼠中的溃疡性结肠炎样疾病
Cell. 1993 Oct 22;75(2):253-61. doi: 10.1016/0092-8674(93)80067-o.
8
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Int Immunol. 1993 Nov;5(11):1461-71. doi: 10.1093/intimm/5.11.1461.
9
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Functions of rat T-lymphocyte subsets isolated by means of monoclonal antibodies.用单克隆抗体分离的大鼠T淋巴细胞亚群的功能。
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