Gialafos J, Maillis A, Kalogeropoulos C, Kalikazaros J, Basiakos L, Avgoustakis D
Am Heart J. 1985 May;109(5 Pt 1):984-91. doi: 10.1016/0002-8703(85)90239-x.
The inhibition of unipolar demand pacemakers by myopotentials was studied in 215 paced patients with or without symptoms and in steady pacing rhythm. ECG recordings were taken of all patients at rest and during effort in which maximal muscular strength from the pectoralis major (PM) and rectus abdominis (RA) muscles was required. In 75 patients (34.9%) transient pacing inhibition was observed. In these patients myopotentials from the PM and RA muscles were recorded simultaneously at rest and during special effort. The PM was the dominant source of inhibiting myopotentials in 50.7% of the total patients with oversensing and the RA was dominant in 28%. In the remaining 21.3%, neither of these muscle groups alone was able to cause inhibition and a clear synergy of both the PM and RA muscles was required. When oversensing occurred in a pacemaker implanted in the thoracic wall, the PM was the dominant cause in 54.4% of patients and the RA in 23.5%, while a synergy of both muscles was required in the remaining 22%. When the pacemaker was implanted in the abdominal wall, the RA was the dominant source of inhibiting myopotentials in all but two patients.
在215例有或无症状且起搏节律稳定的起搏患者中，研究了肌电位对单极按需起搏器的抑制作用。记录了所有患者静息时以及进行需要胸大肌（PM）和腹直肌（RA）产生最大肌肉力量的运动时的心电图。在75例患者（34.9%）中观察到了短暂的起搏抑制。在这些患者中，静息时以及进行特殊运动时同时记录了PM和RA肌肉的肌电位。在所有感知过度的患者中，PM是抑制性肌电位的主要来源，占50.7%，RA占28%。在其余21.3%的患者中，这两组肌肉单独都不能引起抑制，需要PM和RA肌肉明显协同作用。当植入胸壁的起搏器发生感知过度时，54.4%的患者中PM是主要原因，23.5%的患者中RA是主要原因，其余22%的患者需要两组肌肉协同作用。当起搏器植入腹壁时，除两名患者外，RA是抑制性肌电位的主要来源。
