Amikam S, Peleg H, Lemer J, Riss E
Br Heart J. 1977 Nov;39(11):1279-81. doi: 10.1136/hrt.39.11.1279.
A patient is described in whom myopotentials orginating from the anterior abdominal wall muscle suppressed the implanted demand pacemaker despite its bipolar mode of action. This phenomenon was shown by simultaneous recording of the electrocardiogram the electromyogram. At operation, a defect in the insulation of a previously repaired epicardial electrode was found lying in close proximity to these muscles. After repair of the insulation defect, normal pacemaker function was restored. It is suggested that the myopotentials leaked into the pacing system through the insulation defect, thereby suppressing the demand unit, which maintained its bipolar mode of pacing throughout.
本文描述了一名患者，尽管植入的按需起搏器采用双极作用模式，但源自前腹壁肌肉的肌电位仍抑制了该起搏器。通过同步记录心电图和肌电图显示了这种现象。手术时，发现先前修复的心外膜电极绝缘存在缺陷，该电极紧邻这些肌肉。修复绝缘缺陷后，起搏器功能恢复正常。据推测，肌电位通过绝缘缺陷漏入起搏系统，从而抑制了按需起搏器，而该起搏器在整个过程中一直保持其双极起搏模式。
