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Towards precision disease-modelling in experimental myocarditis.迈向实验性心肌炎的精准疾病建模
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Development of a new mouse model for coxsackievirus-induced myocarditis by attenuating coxsackievirus B3 virulence in the pancreas.通过削弱胰腺中柯萨奇病毒 B3 的毒力来开发一种新的柯萨奇病毒诱导心肌炎的小鼠模型。
Cardiovasc Res. 2020 Aug 1;116(10):1756-1766. doi: 10.1093/cvr/cvz259.
3
A non-cardiovirulent strain of coxsackievirus B3 causes myocarditis in mice with severe combined immunodeficiency syndrome.柯萨奇病毒B3的一种非心脏毒性毒株可在患有严重联合免疫缺陷综合征的小鼠中引发心肌炎。
Eur Heart J. 1995 Dec;16 Suppl O:18-9. doi: 10.1093/eurheartj/16.suppl_o.18.
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Transforming growth factor-beta inhibits coxsackievirus-mediated autoimmune myocarditis.转化生长因子-β抑制柯萨奇病毒介导的自身免疫性心肌炎。
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Lack of IL-6 during coxsackievirus infection heightens the early immune response resulting in increased severity of chronic autoimmune myocarditis.柯萨奇病毒感染期间白细胞介素-6的缺乏会增强早期免疫反应，导致慢性自身免疫性心肌炎的严重程度增加。
PLoS One. 2009 Jul 9;4(7):e6207. doi: 10.1371/journal.pone.0006207.
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Autoimmune myocarditis: concepts and questions.自身免疫性心肌炎：概念与问题
Immunol Today. 1991 Aug;12(8):253-5. doi: 10.1016/0167-5699(91)90118-D.
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Group B coxsackievirus myocarditis and pancreatitis: connection between viral virulence phenotypes in mice.B组柯萨奇病毒心肌炎与胰腺炎：小鼠病毒毒力表型之间的联系
J Med Virol. 2000 Sep;62(1):70-81. doi: 10.1002/1096-9071(200009)62:1<70::aid-jmv11>3.0.co;2-r.
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Elution of autoantibodies from the hearts of coxsackievirus-infected mice.从柯萨奇病毒感染小鼠的心脏中洗脱自身抗体。
Eur Heart J. 1991 Aug;12 Suppl D:113-6. doi: 10.1093/eurheartj/12.suppl_d.113.
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An in vivo model of autoimmune post-coxsackievirus B3 myocarditis in severe combined immunodeficiency mouse.
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Toll-like receptor 4-induced cytokine production circumvents protection conferred by TGF-beta in coxsackievirus-mediated autoimmune myocarditis.Toll样受体4诱导的细胞因子产生规避了转化生长因子β在柯萨奇病毒介导的自身免疫性心肌炎中所提供的保护作用。
Clin Immunol. 2006 Dec;121(3):339-49. doi: 10.1016/j.clim.2006.07.009. Epub 2006 Sep 11.

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Targeting regulatory T cells for cardiovascular diseases.靶向调节性 T 细胞治疗心血管疾病。
Front Immunol. 2023 Feb 23;14:1126761. doi: 10.3389/fimmu.2023.1126761. eCollection 2023.

本文引用的文献

1
Development of a new mouse model for coxsackievirus-induced myocarditis by attenuating coxsackievirus B3 virulence in the pancreas.通过削弱胰腺中柯萨奇病毒 B3 的毒力来开发一种新的柯萨奇病毒诱导心肌炎的小鼠模型。
Cardiovasc Res. 2020 Aug 1;116(10):1756-1766. doi: 10.1093/cvr/cvz259.
2
Myocarditis in Humans and in Experimental Animal Models.人类和实验动物模型中的心肌炎
Front Cardiovasc Med. 2019 May 16;6:64. doi: 10.3389/fcvm.2019.00064. eCollection 2019.
3
Complex roads from genotype to phenotype in dilated cardiomyopathy: scientific update from the Working Group of Myocardial Function of the European Society of Cardiology.扩张型心肌病从基因型到表型的复杂途径：欧洲心脏病学会心肌功能工作组的科学更新。
Cardiovasc Res. 2018 Aug 1;114(10):1287-1303. doi: 10.1093/cvr/cvy122.
4
Effusive-constrictive pericarditis, hepatitis, and pancreatitis in a patient with possible coxsackievirus B infection: a case report.一名可能感染柯萨奇病毒B的患者出现渗出性缩窄性心包炎、肝炎和胰腺炎：病例报告
BMC Infect Dis. 2016 Aug 8;16:375. doi: 10.1186/s12879-016-1752-3.
5
Current state of knowledge on aetiology, diagnosis, management, and therapy of myocarditis: a position statement of the European Society of Cardiology Working Group on Myocardial and Pericardial Diseases.心肌炎的病因、诊断、治疗和治疗的现有知识状况：欧洲心脏病学会心肌和心包疾病工作组的立场声明。
Eur Heart J. 2013 Sep;34(33):2636-48, 2648a-2648d. doi: 10.1093/eurheartj/eht210. Epub 2013 Jul 3.
6
Tissue-specific deletion of the coxsackievirus and adenovirus receptor protects mice from virus-induced pancreatitis and myocarditis.柯萨奇病毒和腺病毒受体的组织特异性缺失可保护小鼠免受病毒诱导的胰腺炎和心肌炎。
Cell Host Microbe. 2009 Jul 23;6(1):91-8. doi: 10.1016/j.chom.2009.05.018.
7
Myocarditis: current trends in diagnosis and treatment.心肌炎：诊断与治疗的当前趋势
Circulation. 2006 Feb 14;113(6):876-90. doi: 10.1161/CIRCULATIONAHA.105.584532.
8
Group B coxsackievirus myocarditis and pancreatitis: connection between viral virulence phenotypes in mice.B组柯萨奇病毒心肌炎与胰腺炎：小鼠病毒毒力表型之间的联系
J Med Virol. 2000 Sep;62(1):70-81. doi: 10.1002/1096-9071(200009)62:1<70::aid-jmv11>3.0.co;2-r.
9
Pancreatic expression of interferon-gamma protects mice from lethal coxsackievirus B3 infection and subsequent myocarditis.胰腺中γ干扰素的表达可保护小鼠免受致死性柯萨奇病毒B3感染及随后的心肌炎。
Nat Med. 2000 Jun;6(6):693-7. doi: 10.1038/76277.
10
Cardiac, pancreatic, and liver abnormalities in a patient with coxsackie-B infection.一名柯萨奇B病毒感染患者的心脏、胰腺和肝脏异常。
Am J Med. 1996 Sep;101(3):325-6. doi: 10.1016/S0002-9343(97)89436-3.

Towards precision disease-modelling in experimental myocarditis.

作者信息

Protonotarios Alexandros, Marelli-Berg Federica

机构信息

William Harvey Research Institute, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK.

Institute of Cardiovascular Science, University College London, 72 Huntley Street, London WC1E 6AG, UK.

出版信息

Cardiovasc Res. 2020 Aug 1;116(10):1656-1657. doi: 10.1093/cvr/cvaa057.

DOI:10.1093/cvr/cvaa057
PMID:32167527
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7380721/
Abstract
摘要