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通过Bachmann束单向连接成功消融双房性心动过速并保留左心耳电活动:一例报告

Successful ablation of biatrial tachycardia with preserved electrical activation of left atrial appendage by unidirectional connection via Bachmann's bundle: A case report.

作者信息

Ishiguchi Hironori, Yoshida Masaaki, Ishikura Masahiro, Kawabata Tetsuya, Oda Tsuyoshi

机构信息

Division of Cardiology, Shimane Prefectural Central Hospital, Izumo, Japan.

Division of Cardiology, Shimane Prefectural Central Hospital, Izumo, Japan.

出版信息

Indian Pacing Electrophysiol J. 2020 Jul-Aug;20(4):166-169. doi: 10.1016/j.ipej.2020.04.006. Epub 2020 Apr 17.

Abstract

A 57-year-old man underwent his seventh ablation session for atrial tachycardia (AT). His previous ablations involved several regions of the right atrium (RA) and left atrium (LA). The AT was characterized as biatrial tachycardia with a circuit involving the mitral annulus and septal RA. The AT was terminated by ablation through the insertion site of Bachmann's bundle (BB) in both atria. After 3 months, the patient underwent his eighth ablation session because of AT recurrence. Activation maps showed that the connection from the RA to LA and vice versa was maintained via BB and the coronary sinus, respectively. The ablation target to interrupt the AT circuit was the mitral isthmus (MI), not BB, because BB supplied the electrical activation of the left atrial appendage (LAA) via a unidirectional electrical connection from the RA to LA. Ablation attempts from within the coronary sinus were performed to target the epicardial connection in the MI and led to complete blockage of the connection from the LA to RA. Otherwise, the connection from the RA to LA was preserved via BB. The patient was free of symptoms and anti-arrhythmic drugs at the 4-month follow-up. However, he had a high risk of electrical isolation of the LAA because extensive ablations had been performed; the strategy of targeting the MI contributed to the balance between preserving the electrical activation of the LAA and treating the biatrial tachycardia. Verification of the connective pathway between the two atria might be helpful to determine the optimal target.

摘要

一名57岁男性因房性心动过速(AT)接受了第七次消融治疗。他之前的消融涉及右心房(RA)和左心房(LA)的多个区域。该AT的特征为双房性心动过速,其折返环涉及二尖瓣环和房间隔RA。通过在双心房的巴赫曼束(BB)插入部位进行消融终止了AT。3个月后,患者因AT复发接受了第八次消融治疗。激动标测显示,RA与LA之间的连接分别通过BB和冠状窦得以维持。中断AT折返环的消融靶点是二尖瓣峡部(MI),而非BB,因为BB通过从RA到LA的单向电连接为左心耳(LAA)提供电激动。在冠状窦内进行消融尝试以靶向MI的心外膜连接,导致LA与RA之间的连接完全阻断。否则,RA与LA之间的连接通过BB得以保留。在4个月的随访中,患者无症状且未服用抗心律失常药物。然而,由于已进行了广泛的消融,他存在LAA电隔离的高风险;靶向MI的策略有助于在保留LAA电激动和治疗双房性心动过速之间取得平衡。验证两心房之间的连接途径可能有助于确定最佳靶点。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4064/7384361/642d7e214310/gr1.jpg

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