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本文引用的文献

1
SARS-Coronavirus Open Reading Frame-3a drives multimodal necrotic cell death.严重急性呼吸综合征冠状病毒开放阅读框 3a 驱动多模式坏死性细胞死亡。
Cell Death Dis. 2018 Sep 5;9(9):904. doi: 10.1038/s41419-018-0917-y.
2
Lipopolysaccharide-induced necroptosis of brain microvascular endothelial cells can be prevented by inhibition of endothelin receptors.通过抑制内皮素受体可预防脂多糖诱导的脑微血管内皮细胞坏死性凋亡。
Physiol Res. 2018 Jun 27;67(Suppl 1):S227-S236. doi: 10.33549/physiolres.933842.
3
RIP3: a molecular switch for necrosis and inflammation.RIP3：坏死和炎症的分子开关。
Genes Dev. 2013 Aug 1;27(15):1640-9. doi: 10.1101/gad.223321.113.
4
Many stimuli pull the necrotic trigger, an overview.许多刺激因素引发了细胞坏死，概述如下。
Cell Death Differ. 2012 Jan;19(1):75-86. doi: 10.1038/cdd.2011.164. Epub 2011 Nov 11.
5
Lung pathology of severe acute respiratory syndrome (SARS): a study of 8 autopsy cases from Singapore.严重急性呼吸综合征（SARS）的肺部病理学：对新加坡8例尸检病例的研究
Hum Pathol. 2003 Aug;34(8):743-8. doi: 10.1016/s0046-8177(03)00367-8.

Endothelin receptor antagonists for pulmonary arterial hypertension and COVID-19: Friend or foe?

作者信息

Badagliacca Roberto, Sciomer Susanna, Petrosillo Nicola

机构信息

Department of Cardiovascular and Respiratory Science, Sapienza University of Rome, Rome, Italy.

Department of Cardiovascular and Respiratory Science, Sapienza University of Rome, Rome, Italy.

出版信息

J Heart Lung Transplant. 2020 Jul;39(7):729-730. doi: 10.1016/j.healun.2020.04.007. Epub 2020 Apr 17.

DOI:10.1016/j.healun.2020.04.007
PMID:32360293
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7162760/
Abstract
摘要