肾素-血管紧张素-醛固酮系统的激活与 COVID-19 中的急性肾损伤有关。
Activation of the renin-angiotensin-aldosterone system is associated with Acute Kidney Injury in COVID-19.
机构信息
Assistance Publique-Hôpitaux de Paris (AP-HP), Groupe Hospitalier St Louis-Lariboisière, Department of Anaesthesiology and Critical Care and Burn Unit, Paris, France; University of Paris, Paris, France.
Assistance Publique-Hôpitaux de Paris (AP-HP), Groupe Hospitalier St Louis-Lariboisière, Department of Biochemistry, Paris, France.
出版信息
Anaesth Crit Care Pain Med. 2020 Aug;39(4):453-455. doi: 10.1016/j.accpm.2020.06.006. Epub 2020 Jun 18.
The pathophysiology of acute kidney injury (AKI) in COVID-19 patients is still poorly understood. SARS-CoV-2 has been suggested to modulate the renin-angiotensin-aldosterone system (RAAS). In this series of COVID-19 critically ill patients, we report evidence of activation of the RAAS in COVID-19 patients with AKI.
COVID-19 患者急性肾损伤（AKI）的病理生理学仍知之甚少。有人提出，SARS-CoV-2 可调节肾素-血管紧张素-醛固酮系统（RAAS）。在这一系列 COVID-19 危重症患者中，我们报告了 COVID-19 合并 AKI 患者 RAAS 激活的证据。
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