产生颗粒酶B的皮肤T细胞与斑秃疾病预后的可能联系。 - Suppr | 超能文献

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The Possible Linkage of Granzyme B-Producing Skin T Cells with the Disease Prognosis of Alopecia Areata.产生颗粒酶B的皮肤T细胞与斑秃疾病预后的可能联系。

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Analysis of the expression of cutaneous lymphocyte-associated antigen on the peripheral blood and cutaneous lymphocytes of alopecia areata patients.斑秃患者外周血及皮肤淋巴细胞中皮肤淋巴细胞相关抗原表达的分析

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Resistance to alopecia areata in C3H/HeJ mice is associated with increased expression of regulatory cytokines and a failure to recruit CD4+ and CD8+ cells.C3H/HeJ小鼠对斑秃的抗性与调节性细胞因子表达增加以及无法募集CD4+和CD8+细胞有关。

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Mediation of alopecia areata by cooperation between CD4+ and CD8+ T lymphocytes: transfer to human scalp explants on Prkdc(scid) mice.CD4+和CD8+ T淋巴细胞之间的合作介导斑秃：移植到Prkdc(scid)小鼠的人头皮外植体上。

Arch Dermatol. 2002 Jul;138(7):916-22. doi: 10.1001/archderm.138.7.916.

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Increase in CD8 Effector Memory T Cells Re-Expressing CD45RA Correlates with Intractability of Severe Alopecia Areata.重新表达CD45RA的CD8效应记忆T细胞增加与重度斑秃的难治性相关。

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Cellular retinol-binding protein-1 expression increases with increasing clinical severity of alopecia areata.细胞视黄醇结合蛋白-1的表达随着斑秃临床严重程度的增加而升高。

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Comparison of interleukin-17- producing cells in different clinical types of alopecia areata.不同临床类型斑秃患者中白细胞介素 17 产生细胞的比较。

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The Evolving Pathogenesis of Alopecia Areata: Major Open Questions.斑秃不断演变的发病机制：主要悬而未决的问题。

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Perinevoid alopecia: a case report and literature review.阴部无瘢痕性脱发：一例报告及文献综述

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Substance P as an immunomodulatory neuropeptide in a mouse model for autoimmune hair loss (alopecia areata).在自身免疫性脱发（斑秃）小鼠模型中，P物质作为一种免疫调节性神经肽。

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Deciphering the Complex Immunopathogenesis of Alopecia Areata.解析斑秃的复杂免疫发病机制。

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Granzyme B in Autoimmune Skin Disease.颗粒酶 B 与自身免疫性皮肤病。

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Skin Interstitial Fluid and Plasma Multiplex Cytokine Analysis Reveals IFN-γ Signatures and Granzyme B as Useful Biomarker for Activity, Severity and Prognosis Assessment in Vitiligo.皮肤细胞间液和血浆多重细胞因子分析揭示 IFN-γ 特征和颗粒酶 B 可作为评估白癜风活动性、严重程度和预后的有用生物标志物。