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享乐性饥饿作为门诊认知行为治疗神经性贪食症的作用机制。

Hedonic hunger as a mechanism of action in outpatient cognitive behavioral therapy for bulimia nervosa.

机构信息

Department of Psychology, Drexel University, Philadelphia, Pennsylvania, USA.

出版信息

Int J Eat Disord. 2020 Sep;53(9):1539-1543. doi: 10.1002/eat.23339. Epub 2020 Jul 7.

Abstract

OBJECTIVE

Hedonic hunger (i.e., the motivation to consume palatable foods in the absence of an energy deficit) has been associated with the onset and maintenance of loss of control (LOC) eating. However, it remains underexplored as a mechanism of action in outpatient cognitive behavioral therapy (CBT) for bulimia nervosa (BN). In the present study, we hypothesized that reductions in hedonic hunger would significantly mediate reductions in overall eating pathology and LOC episodes in two samples (N = 28, N = 23) of 20 and 16 sessions, respectively.

METHOD

Participants completed the Eating Disorder Examination (EDE) and Power of Food Scale (PFS) at pre- and post-treatment.

RESULTS

In both samples, EDE Global scores, LOC episodes, and PFS Total scores significantly improved over the course of treatment. In Sample 1, significant indirect effects of PFS Total scores on EDE Global scores and LOC episodes were observed. In Sample 2, the indirect effect of PFS Total scores was significant on EDE Global scores and nonsignificant on LOC episodes though it followed a similar pattern of change.

DISCUSSION

Results suggest that reductions in hedonic hunger are associated with better outcomes in CBT for BN. Replication and further research is needed to elucidate the treatment components driving these reductions.

摘要

目的

享乐性饥饿(即在不存在能量不足的情况下进食美味食物的动机)与失控(LOC)进食的发生和维持有关。然而,作为门诊认知行为疗法(CBT)治疗神经性贪食症(BN)的作用机制,它仍未得到充分探索。在本研究中,我们假设在两个分别为 20 次和 16 次治疗的样本(N=28,N=23)中,享乐性饥饿感的减少将显著介导总体饮食病理学和 LOC 发作的减少。

方法

参与者在治疗前和治疗后完成饮食障碍检查(EDE)和食物力量量表(PFS)。

结果

在两个样本中,EDE 全球评分、LOC 发作和 PFS 总分在治疗过程中均显著改善。在样本 1 中,PFS 总分对 EDE 全球评分和 LOC 发作的间接效应显著。在样本 2 中,PFS 总分对 EDE 全球评分的间接效应显著,而对 LOC 发作的间接效应不显著,但变化模式相似。

讨论

结果表明,享乐性饥饿感的减少与 CBT 治疗 BN 的更好结果相关。需要进一步的复制和研究来阐明驱动这些减少的治疗成分。

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