• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

论与 COVID-19 大流行相关的迟发性神经精神表现的可能性。

On the plausibility of late neuropsychiatric manifestations associated with the COVID-19 pandemic.

机构信息

Neurology Department, Algarve University Hospital, Portugal.

Psychiatry Department, Algarve University Hospital, Portugal.

出版信息

J Neurol Sci. 2020 Oct 15;417:117060. doi: 10.1016/j.jns.2020.117060. Epub 2020 Jul 27.

DOI:10.1016/j.jns.2020.117060
PMID:32739501
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7384401/
Abstract

• Virus-host interactions in COVID-19 pandemic is a substrate for the emergence of late neurological manifestations. • Occurrence subacute autoimmune disorders after COVID-19 suggests the risk of late neurological autoimmune disturbance. • Global monitoring is important to uncover emergence of late neuropsychiatric manifestations.

摘要

• COVID-19 大流行中的病毒-宿主相互作用是晚期神经表现出现的基础。 • COVID-19 后发生亚急性自身免疫性疾病表明存在晚期神经自身免疫紊乱的风险。 • 全球监测对于揭示晚期神经精神表现的出现很重要。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1167/7384401/b0d693ca8c72/gr1_lrg.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1167/7384401/b0d693ca8c72/gr1_lrg.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/1167/7384401/b0d693ca8c72/gr1_lrg.jpg

相似文献

1
On the plausibility of late neuropsychiatric manifestations associated with the COVID-19 pandemic.论与 COVID-19 大流行相关的迟发性神经精神表现的可能性。
J Neurol Sci. 2020 Oct 15;417:117060. doi: 10.1016/j.jns.2020.117060. Epub 2020 Jul 27.
2
Neuro-COVID-19: What are we talking about?神经新冠病毒感染：我们在讨论什么？
Rev Neurol (Paris). 2020 Sep;176(7-8):527-528. doi: 10.1016/j.neurol.2020.05.004. Epub 2020 Jun 6.
3
Are we facing a crashing wave of neuropsychiatric sequelae of COVID-19? Neuropsychiatric symptoms and potential immunologic mechanisms.我们是否正面临着一波 COVID-19 神经精神后遗症的浪潮？神经精神症状和潜在的免疫机制。
Brain Behav Immun. 2020 Jul;87:34-39. doi: 10.1016/j.bbi.2020.04.027. Epub 2020 Apr 13.
4
The neurology of COVID-19 revisited: A proposal from the Environmental Neurology Specialty Group of the World Federation of Neurology to implement international neurological registries.重新审视 COVID-19 相关神经学：世界神经病学联合会环境神经病学专业组提出实施国际神经学登记的建议。
J Neurol Sci. 2020 Jul 15;414:116884. doi: 10.1016/j.jns.2020.116884. Epub 2020 May 7.
5
Impact of SARS-CoV-2 infection on neurodegenerative and neuropsychiatric diseases: a delayed pandemic?新型冠状病毒2型感染对神经退行性疾病和神经精神疾病的影响：一场延迟的大流行？
Neurologia (Engl Ed). 2020 May;35(4):245-251. doi: 10.1016/j.nrl.2020.04.002. Epub 2020 Apr 17.
6
Understanding pathophysiology of hemostasis disorders in critically ill patients with COVID-19.了解新冠肺炎危重症患者止血障碍的病理生理学。
Intensive Care Med. 2020 Aug;46(8):1603-1606. doi: 10.1007/s00134-020-06088-1. Epub 2020 May 15.
7
Understanding the Immunologic Characteristics of Neurologic Manifestations of SARS-CoV-2 and Potential Immunological Mechanisms.了解 SARS-CoV-2 神经系统表现的免疫学特征和潜在的免疫学机制。
Mol Neurobiol. 2020 Dec;57(12):5263-5275. doi: 10.1007/s12035-020-02094-y. Epub 2020 Sep 1.
8
COVID-19 and myocardial injury: is there a role for interleukin-1 inhibition?2019冠状病毒病与心肌损伤：白细胞介素-1抑制是否起作用？
J Cardiovasc Med (Hagerstown). 2020 Jul;21(7):465-466. doi: 10.2459/JCM.0000000000000998.
9
Immune-Mediated Coagulopathy in COVID-19 Infection.新型冠状病毒肺炎感染中的免疫介导性凝血病
Semin Thromb Hemost. 2020 Oct;46(7):838-840. doi: 10.1055/s-0040-1714272. Epub 2020 Sep 2.
10
Cellular immune responses to covid-19.对新冠病毒的细胞免疫反应。
BMJ. 2020 Jul 31;370:m3018. doi: 10.1136/bmj.m3018.

引用本文的文献

1
The Impact of COVID-19 Infection and Enforced Prolonged Social Isolation on Neuropsychiatric Symptoms in Older Adults With and Without Dementia: A Review.新冠病毒感染及强制长期社会隔离对有无痴呆症老年人神经精神症状的影响：一项综述
Front Psychiatry. 2020 Oct 22;11:585540. doi: 10.3389/fpsyt.2020.585540. eCollection 2020.

本文引用的文献

1
Molecular mimicry may explain multi-organ damage in COVID-19.分子模拟或许可以解释新冠病毒感染中多器官损伤的现象。
Autoimmun Rev. 2020 Aug;19(8):102591. doi: 10.1016/j.autrev.2020.102591. Epub 2020 Jun 11.
2
Neuropathological Features of Covid-19.新冠病毒病的神经病理学特征
N Engl J Med. 2020 Sep 3;383(10):989-992. doi: 10.1056/NEJMc2019373. Epub 2020 Jun 12.
3
Neuropathogenesis and Neurologic Manifestations of the Coronaviruses in the Age of Coronavirus Disease 2019: A Review.新型冠状病毒病 2019 时代冠状病毒的神经发病机制和神经表现：综述。
JAMA Neurol. 2020 Aug 1;77(8):1018-1027. doi: 10.1001/jamaneurol.2020.2065.
4
Guillain Barre syndrome associated with COVID-19 infection: A case report.与新型冠状病毒肺炎感染相关的吉兰-巴雷综合征：一例报告
J Clin Neurosci. 2020 Jun;76:233-235. doi: 10.1016/j.jocn.2020.04.062. Epub 2020 Apr 15.
5
Central nervous system manifestations of COVID-19: A systematic review.COVID-19 相关的中枢神经系统表现：一项系统性综述。
J Neurol Sci. 2020 Jun 15;413:116832. doi: 10.1016/j.jns.2020.116832. Epub 2020 Apr 11.
6
Genetic diversity and evolution of SARS-CoV-2.SARS-CoV-2 的遗传多样性与演化。
Infect Genet Evol. 2020 Jul;81:104260. doi: 10.1016/j.meegid.2020.104260. Epub 2020 Feb 21.
7
Active HHV-6 Infection of Cerebellar Purkinje Cells in Mood Disorders.情绪障碍中小脑浦肯野细胞的HHV-6活动性感染
Front Microbiol. 2018 Aug 21;9:1955. doi: 10.3389/fmicb.2018.01955. eCollection 2018.
8
Primary Psychiatric Manifestations of Subacute Sclerosing Panencephalitis: A Case Report and Literature Review.
Psychosomatics. 2018 Jul-Aug;59(4):408-412. doi: 10.1016/j.psym.2017.12.004. Epub 2017 Dec 27.
9
Encephalitis lethargica: 100 years after the epidemic.昏睡性脑炎：疫情爆发100年后。
Brain. 2017 Aug 1;140(8):2246-2251. doi: 10.1093/brain/awx177.
10
Association of seropositivity for influenza and coronaviruses with history of mood disorders and suicide attempts.流感和冠状病毒血清阳性与心境障碍和自杀未遂史的关联。
J Affect Disord. 2011 Apr;130(1-2):220-5. doi: 10.1016/j.jad.2010.09.029. Epub 2010 Oct 27.