Jiresová M, Janecek J
Folia Microbiol (Praha). 1977;22(3):173-81. doi: 10.1007/BF02885598.
Infection of Escherichia cooi with T1, T2r+, T3 and T4 phages leads to an immediate inhibition of beta-galactosidase synthesis. Similar results were obtained with the virulent mutant of phage lambda. The degree of inhibition of beta-galactosidase synthesis depends on the time delay between the addition of the inducer and the phage particles, and on the amount of phage DNA, which has penetrated into the host cell. RNA phage MS2 exhibited no inhibitory effect on enzyme synthesis.
大肠杆菌被T1、T2r +、T3和T4噬菌体感染会导致β-半乳糖苷酶合成立即受到抑制。用λ噬菌体的烈性突变体也得到了类似的结果。β-半乳糖苷酶合成的抑制程度取决于诱导剂添加与噬菌体颗粒添加之间的时间间隔，以及已渗透到宿主细胞中的噬菌体DNA的量。RNA噬菌体MS2对酶的合成没有抑制作用。
