Chlorine Gas, Airway Inflammation, and Cysteinyl Leukotrienes: The Neutrophil Does Not Work Alone.
作者信息
Sporn Peter H S
机构信息
Division of Pulmonary and Critical Care Medicine, Northwestern University, Chicago, Illinois.
出版信息
Am J Respir Cell Mol Biol. 2020 Nov;63(5):553-554. doi: 10.1165/rcmb.2020-0347ED.
Abstract
摘要
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本文引用的文献
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Montelukast reduces inhaled chlorine triggered airway hyperresponsiveness and airway inflammation in the mouse.孟鲁司特可降低小鼠吸入氯气诱发的气道高反应性和气道炎症。
Br J Pharmacol. 2017 Oct;174(19):3346-3358. doi: 10.1111/bph.13953. Epub 2017 Aug 23.
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CysLT1 Receptor Is Protective against Oxidative Stress in a Model of Irritant-Induced Asthma.半胱氨酰白三烯1受体在刺激性诱导哮喘模型中对氧化应激具有保护作用。
J Immunol. 2016 Jul 1;197(1):266-77. doi: 10.4049/jimmunol.1501084. Epub 2016 May 25.
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Neutrophils mediate airway hyperresponsiveness after chlorine-induced airway injury in the mouse.中性粒细胞介导氯诱导的气道损伤后小鼠气道高反应性。
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