Barss V A, Doubilet P M, St John-Sutton M, Cartier M S, Frigoletto F D
Obstet Gynecol. 1987 Sep;70(3 Pt 2):442-4.
Pulsed Doppler studies of left and right ventricular outputs were obtained over time in a hydropic fetus with erythroblastosis fetalis. Despite severe anemia, cardiac outputs were within the normal range and remained normal after in utero percutaneous intravascular transfusions, which reversed the hydrops. The measurement of cardiac output in utero provides direct evidence that high-output failure due to anemia is not the mechanism for hydrops in erythroblastosis fetalis.
对一名患有胎儿水肿型胎儿红细胞增多症的水肿胎儿，随时间进行了左右心室输出量的脉冲多普勒研究。尽管存在严重贫血，但心输出量仍在正常范围内，并且在宫内经皮血管内输血使水肿消退后仍保持正常。宫内心输出量的测量提供了直接证据，表明贫血导致的高输出量衰竭不是胎儿水肿型胎儿红细胞增多症中水肿的发生机制。
