Department of Radiology, University of Washington School of Medicine, Seattle Children's Hospital, 4800 Sand Point Way NE, Seattle, WA, 98105, USA.
Department of Neurology, University of Washington School of Medicine, Seattle Children's Hospital, Seattle, WA, USA.
Pediatr Radiol. 2021 May;51(5):675-685. doi: 10.1007/s00247-020-04846-3. Epub 2020 Oct 22.
Perinatal venous stroke has classically been attributed to cerebral sinovenous thrombosis with resultant congestion or thrombosis of the small veins draining the cerebrum. Advances in brain MRI, in particular susceptibility-weighted imaging, have enabled the visualization of the engorged small intracerebral veins, and the spectrum of perinatal venous stroke has expanded to include isolated congestion or thrombosis of the deep medullary veins and the superficial intracerebral veins. Congestion or thrombosis of the deep medullary veins or the superficial intracerebral veins can result in vasogenic edema, cytotoxic edema or hemorrhage in the territory of disrupted venous flow. Deep medullary vein engorgement and superficial medullary vein engorgement have characteristic findings on MRI and should be differentiated from neonatal hemorrhagic stroke.
围产期静脉性卒中通常归因于脑静脉窦血栓形成,导致大脑引流的小静脉充血或血栓形成。脑 MRI 的进步,特别是磁敏感加权成像,使人们能够观察到充盈的颅内小静脉,围产期静脉性卒中的范围已经扩大到包括孤立的深部髓静脉和浅部脑静脉充血或血栓形成。深部髓静脉或浅部脑静脉的充血或血栓形成可导致受破坏静脉血流区域的血管源性水肿、细胞毒性水肿或出血。深部髓静脉充盈和浅部髓静脉充盈在 MRI 上具有特征性表现,应与新生儿出血性卒中相区别。