文献检索

Institute of Physiology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health (BIH), Berlin, Germany.

Dept of Infectious Diseases and Respiratory Medicine, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health (BIH), Berlin, Germany.

出版信息

Eur Respir J. 2021 Mar 11;57(3). doi: 10.1183/13993003.02384-2020. Print 2021 Mar.

DOI:10.1183/13993003.02384-2020

PMID:33154030

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7651836/

相似文献

1

Plasma mediators in patients with severe COVID-19 cause lung endothelial barrier failure.严重 COVID-19 患者的血浆介质导致肺血管内皮屏障功能衰竭。

Eur Respir J. 2021 Mar 11;57(3). doi: 10.1183/13993003.02384-2020. Print 2021 Mar.

2

Elevated Cytokine Levels in Plasma of Patients with SARS-CoV-2 Do Not Contribute to Pulmonary Microvascular Endothelial Permeability.SARS-CoV-2 感染者血浆细胞因子水平升高不会导致肺微血管内皮通透性增加。

Microbiol Spectr. 2022 Feb 23;10(1):e0167121. doi: 10.1128/spectrum.01671-21. Epub 2022 Feb 16.

3

Lipolysis products from triglyceride-rich lipoproteins increase endothelial permeability, perturb zonula occludens-1 and F-actin, and induce apoptosis.富含甘油三酯的脂蛋白产生的脂解产物会增加内皮通透性，扰乱紧密连接蛋白-1和F-肌动蛋白，并诱导细胞凋亡。

Am J Physiol Heart Circ Physiol. 2007 Jun;292(6):H2745-53. doi: 10.1152/ajpheart.00686.2006. Epub 2007 Jan 26.

4

Molecular mechanisms mediating protective effect of cAMP on lipopolysaccharide (LPS)-induced human lung microvascular endothelial cells (HLMVEC) hyperpermeability.介导环磷酸腺苷（cAMP）对脂多糖（LPS）诱导的人肺微血管内皮细胞（HLMVEC）高通透性保护作用的分子机制。

J Cell Physiol. 2009 Dec;221(3):750-9. doi: 10.1002/jcp.21913.

5

Impaired integrin-mediated adhesion contributes to reduced barrier properties in VASP-deficient microvascular endothelium.整合素介导的黏附受损导致VASP缺陷型微血管内皮细胞的屏障特性降低。

J Cell Physiol. 2009 Aug;220(2):357-66. doi: 10.1002/jcp.21772.

6

Endothelial cell Piezo1 mediates pressure-induced lung vascular hyperpermeability via disruption of adherens junctions.内皮细胞 Piezo1 通过破坏黏附连接介导压力诱导的肺血管通透性增加。

Proc Natl Acad Sci U S A. 2019 Jun 25;116(26):12980-12985. doi: 10.1073/pnas.1902165116. Epub 2019 Jun 11.

7

HIV-1 Tat C phosphorylates VE-cadherin complex and increases human brain microvascular endothelial cell permeability.HIV-1反式激活因子C使血管内皮钙黏蛋白复合体磷酸化并增加人脑微血管内皮细胞的通透性。

BMC Neurosci. 2014 Jun 26;15:80. doi: 10.1186/1471-2202-15-80.

8

Rap1 Small GTPase Regulates Vascular Endothelial-Cadherin-Mediated Endothelial Cell-Cell Junctions and Vascular Permeability.Rap1小GTP酶调节血管内皮钙黏蛋白介导的内皮细胞间连接和血管通透性。

Biol Pharm Bull. 2021;44(10):1371-1379. doi: 10.1248/bpb.b21-00504.

9

Vasodilator-stimulated phosphoprotein deficiency potentiates PAR-1-induced increase in endothelial permeability in mouse lungs.血管扩张刺激磷蛋白缺乏可增强 PAR-1 诱导的小鼠肺内皮通透性增加。

J Cell Physiol. 2011 May;226(5):1255-64. doi: 10.1002/jcp.22453.

10

HIV-1 Tat C modulates expression of miRNA-101 to suppress VE-cadherin in human brain microvascular endothelial cells.HIV-1 Tat C 调节 miRNA-101 的表达，抑制人脑微血管内皮细胞中的 VE-钙黏蛋白。

J Neurosci. 2013 Apr 3;33(14):5992-6000. doi: 10.1523/JNEUROSCI.4796-12.2013.

引用本文的文献

1

CXCL10-dependent epithelial-vascular cross-talk for endothelial activation following SARS-CoV-2 infection.SARS-CoV-2感染后CXCL10依赖性上皮-血管相互作用促进内皮细胞活化

Sci Rep. 2025 Jul 1;15(1):21129. doi: 10.1038/s41598-025-08329-z.

2

Advances in acute respiratory distress syndrome: focusing on heterogeneity, pathophysiology, and therapeutic strategies.急性呼吸窘迫综合征的进展：聚焦于异质性、病理生理学和治疗策略。

Signal Transduct Target Ther. 2025 Mar 7;10(1):75. doi: 10.1038/s41392-025-02127-9.

3

SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells.

严重 COVID-19 患者的血浆介质导致肺血管内皮屏障功能衰竭。

Plasma mediators in patients with severe COVID-19 cause lung endothelial barrier failure.

机构信息

Institute of Physiology, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health (BIH), Berlin, Germany.

Dept of Infectious Diseases and Respiratory Medicine, Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health (BIH), Berlin, Germany.