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Process of foam cell formation in diet-induced hypercholesterolemic rabbit and the Watanabe heritable hyperlipidemic rabbit.

作者信息

Ikeda M, Kodama H, Nohara N

出版信息

J Dermatol. 1987 Aug;14(4):305-12. doi: 10.1111/j.1346-8138.1987.tb03583.x.

DOI:10.1111/j.1346-8138.1987.tb03583.x
PMID:3320124
Abstract
摘要

相似文献

1
Process of foam cell formation in diet-induced hypercholesterolemic rabbit and the Watanabe heritable hyperlipidemic rabbit.饮食诱导的高胆固醇血症兔和渡边遗传性高脂血症兔中泡沫细胞形成的过程。
J Dermatol. 1987 Aug;14(4):305-12. doi: 10.1111/j.1346-8138.1987.tb03583.x.
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Foam cell formation containing lipid droplets enriched with free cholesterol by hyperlipidemic serum.高脂血症血清诱导富含游离胆固醇的脂滴形成泡沫细胞。
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Fatty streak expansion and maturation in Watanabe Heritable Hyperlipemic and comparably hypercholesterolemic fat-fed rabbits.渡边遗传性高脂血症兔和高脂喂养的高胆固醇血症兔中脂肪条纹的扩展与成熟
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Cholesterol oxidation products induce vascular foam cell lesion formation in hypercholesterolemic New Zealand white rabbits.
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Fatty streak initiation in Watanabe Heritable Hyperlipemic and comparably hypercholesterolemic fat-fed rabbits.
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Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic mice.通过清道夫受体A或CD36途径的受体介导的脂质摄取丧失并不能改善高脂血症小鼠的动脉粥样硬化。
J Clin Invest. 2005 Aug;115(8):2192-201. doi: 10.1172/JCI24061.
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Uptake of acetylated LDL by peritoneal macrophages obtained from normal and Watanabe heritable hyperlipidemic rabbits, an animal model for familial hypercholesterolemia.从正常和渡边遗传性高脂血症兔(一种家族性高胆固醇血症的动物模型)获取的腹膜巨噬细胞对乙酰化低密度脂蛋白的摄取。
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Correlation between the presence of an immature smooth muscle cell population in tunica media and the development of atherosclerotic lesion. A study on different-sized rabbit arteries from cholesterol-fed and Watanabe heritable hyperlipemic rabbits.中膜中未成熟平滑肌细胞群的存在与动脉粥样硬化病变发展之间的相关性。对喂食胆固醇的兔子和渡边遗传性高脂血症兔子不同大小动脉的研究。
Atherosclerosis. 1995 Jul;116(1):77-92. doi: 10.1016/0021-9150(95)05530-a.

引用本文的文献

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The History of the WHHL Rabbit, an Animal Model of Familial Hypercholesterolemia (I) - Contribution to the Elucidation of the Pathophysiology of Human Hypercholesterolemia and Coronary Heart Disease.载脂蛋白 E 基因缺陷兔模型的建立及其在动脉粥样硬化研究中的应用
J Atheroscler Thromb. 2020 Feb 1;27(2):105-118. doi: 10.5551/jat.RV17038-1. Epub 2019 Nov 21.
2
Enhancement of the antigen-presenting function of monocytes by cholesterol: possible relevance to inflammatory mechanisms in extrinsic allergic alveolitis and atherosclerosis.胆固醇增强单核细胞的抗原呈递功能:与外源性过敏性肺泡炎和动脉粥样硬化炎症机制的可能关联。
Clin Exp Immunol. 1992 Feb;87(2):279-86. doi: 10.1111/j.1365-2249.1992.tb02988.x.