Cannabinoid pharmacology and its therapeutic uses in Alzheimer's disease.

作者信息

Monteiro Kadja Luana Chagas, Dos Santos Alcântara Marcone Gomes, de Aquino Thiago Mendonça, da Silva-Júnior Edeildo Ferreira

机构信息

Laboratory of Medicinal Chemistry, Institute of Pharmaceutical Sciences, Federal University of Alagoas, Maceió, Brazil.

Chemistry and Biotechnology Institute, Federal University of Alagoas, Maceió, Brazil.

出版信息

Neural Regen Res. 2021 May;16(5):990-991. doi: 10.4103/1673-5374.294336.

DOI:10.4103/1673-5374.294336

PMID:33229747

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8178768/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/49c8/8178768/b5b4344794ce/NRR-16-990-g001.jpg

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本文引用的文献

[1]

Overview of cannabidiol (CBD) and its analogues: Structures, biological activities, and neuroprotective mechanisms in epilepsy and Alzheimer's disease.

Eur J Med Chem. 2020-2-22

[2]

Cannabidiol: Recent advances and new insights for neuropsychiatric disorders treatment.

Life Sci. 2019-3-22

[3]

Cannabidiol Modulates the Expression of Alzheimer's Disease-Related Genes in Mesenchymal Stem Cells.

Int J Mol Sci. 2016-12-23

[4]

The potential therapeutic effects of THC on Alzheimer's disease.

J Alzheimers Dis. 2014

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Cannabinoid effects on β amyloid fibril and aggregate formation, neuronal and microglial-activated neurotoxicity in vitro.

Cell Mol Neurobiol. 2013-9-13

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Natural cannabinoids improve dopamine neurotransmission and tau and amyloid pathology in a mouse model of tauopathy.

J Alzheimers Dis. 2013

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Cannabidiol reduces Aβ-induced neuroinflammation and promotes hippocampal neurogenesis through PPARγ involvement.

PLoS One. 2011-12-5

[8]

Cannabidiol and other cannabinoids reduce microglial activation in vitro and in vivo: relevance to Alzheimer's disease.

Mol Pharmacol. 2011-2-24

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Endocannabinoid 2-arachidonoylglycerol protects neurons against β-amyloid insults.

Neuroscience. 2011-1-19

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CB1 receptor selective activation inhibits beta-amyloid-induced iNOS protein expression in C6 cells and subsequently blunts tau protein hyperphosphorylation in co-cultured neurons.

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