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利用肠道微生物组治疗肝硬化失代偿期和慢加急性肝衰竭。

Utilizing the gut microbiome in decompensated cirrhosis and acute-on-chronic liver failure.

机构信息

Translational Hepatology, Department of Internal Medicine I, Goethe University Clinic Frankfurt, Frankfurt, Germany.

European Foundation for the Study of Chronic Liver Failure, Barcelona, Spain.

出版信息

Nat Rev Gastroenterol Hepatol. 2021 Mar;18(3):167-180. doi: 10.1038/s41575-020-00376-3. Epub 2020 Nov 30.

Abstract

The human gut microbiome has emerged as a major player in human health and disease. The liver, as the first organ to encounter microbial products that cross the gut epithelial barrier, is affected by the gut microbiome in many ways. Thus, the gut microbiome might play a major part in the development of liver diseases. The common end stage of liver disease is decompensated cirrhosis and the further development towards acute-on-chronic liver failure (ACLF). These conditions have high short-term mortality. There is evidence that translocation of components of the gut microbiota, facilitated by different pathogenic mechanisms such as increased gut epithelial permeability and portal hypertension, is an important driver of decompensation by induction of systemic inflammation, and thereby also ACLF. Elucidating the role of the gut microbiome in the aetiology of decompensated cirrhosis and ACLF deserves further investigation and improvement; and might be the basis for development of diagnostic and therapeutic strategies. In this Review, we focus on the possible pathogenic, diagnostic and therapeutic role of the gut microbiome in decompensation of cirrhosis and progression to ACLF.

摘要

人类肠道微生物组已成为人类健康和疾病的主要参与者。肝脏作为第一个遇到穿过肠道上皮屏障的微生物产物的器官,受到肠道微生物组的多方面影响。因此,肠道微生物组可能在肝脏疾病的发展中起主要作用。肝脏疾病的常见终末期是失代偿性肝硬化,进一步发展为慢加急性肝衰竭(ACLF)。这些情况具有很高的短期死亡率。有证据表明,肠道微生物群的组成部分通过不同的发病机制易位,如增加肠道上皮通透性和门静脉高压,通过诱导全身炎症是失代偿的重要驱动因素,从而也是 ACLF 的重要驱动因素。阐明肠道微生物组在失代偿性肝硬化和 ACLF 发病机制中的作用值得进一步研究和改善;并可能成为开发诊断和治疗策略的基础。在这篇综述中,我们重点讨论了肠道微生物组在肝硬化失代偿和进展为 ACLF 中的可能致病、诊断和治疗作用。

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