Faculty of Health Science, European University of Canarias, Tenerife, Spain.
Department of Physical Education and Sport, Faculty of Sport Sciences, Catholic University of Murcia (UCAM), Murcia, Spain.
J Strength Cond Res. 2021 Apr 1;35(4):886-893. doi: 10.1519/JSC.0000000000003921.
González-Hernández, JM, García-Ramos, A, Colomer-Poveda, D, Tvarijonaviciute, A, Cerón, J, Jiménez-Reyes, P, and Márquez, G. Resistance training to failure vs. not to failure: acute and delayed markers of mechanical, neuromuscular, and biochemical fatigue. J Strength Cond Res 35(4): 886-893, 2021-This study aimed to compare acute and delayed markers of mechanical, neuromuscular, and biochemical fatigue between resistance training sessions leading to or not to failure. Twelve resistance-trained men completed 2 sessions that consisted of 6 sets of the full-squat exercise performed against the 10 repetitions maximum load. In a randomized order, in one session the sets were performed to failure and in the other session the sets were not performed to failure (5 repetitions per set). Mechanical fatigue was quantified through the recording of the mean velocity during all repetitions. The neuromuscular function of the knee extensors was assessed through a maximal voluntary contraction and the twitch interpolation technique before training, immediately after each set, and 1, 24, and 48 hours post-training. Serum creatine kinase (CK) and aspartate aminotransferase (AST) were measured before training and 1, 24, and 48 hours post-training to infer muscle damage. Alpha was set at a level of 0.05. A higher velocity loss between sets was observed during the failure protocol (-21.7%) compared with the nonfailure protocol (-3.5%). The markers of peripheral fatigue were generally higher and long lasting for the failure protocol. However, the central fatigue assessed by the voluntary activation was comparable for both protocols and remained depressed up to 48 hours post-training. The concentrations of CK and AST were higher after the failure protocol revealing higher muscle damage compared with the nonfailure protocol. These results support the nonfailure protocol to reduce peripheral fatigue and muscle damage, whereas the central fatigue does not seem to be affected by the set configuration.
冈萨雷斯 - 埃尔南德斯，JM，加西亚 - 拉莫斯，A，科洛默尔 - 波韦达，D，塔维乔纳维丘特，A，塞隆，J，希门尼斯 - 雷耶斯，P，和马尔克斯，G。失败与非失败的抗阻训练：机械、神经肌肉和生化疲劳的急性和延迟标志物。J 力量与调节研究 35(4)：886-893，2021-本研究旨在比较导致或不导致失败的抗阻训练课之间的机械、神经肌肉和生化疲劳的急性和延迟标志物。12 名受过抗阻训练的男性完成了 2 次训练，包括 6 组全蹲练习，每组重复 10 次最大负荷。在随机顺序中，在一次训练中，每组进行至力竭，而在另一次训练中，每组不进行至力竭（每组 5 次重复）。通过记录所有重复过程中的平均速度来量化机械疲劳。在训练前、每次训练后即刻以及训练后 1、24 和 48 小时，通过最大自主收缩和肌电图技术评估膝关节伸肌的神经肌肉功能。在训练前和训练后 1、24 和 48 小时测量血清肌酸激酶（CK）和天冬氨酸氨基转移酶（AST），以推断肌肉损伤。设定 alpha 值为 0.05。在失败方案中，各组之间的速度损失更高（-21.7%），而在非失败方案中（-3.5%）。对于失败方案，周围性疲劳标志物通常更高且持续时间更长。然而，两种方案的自愿激活评估的中枢疲劳相当，并且在训练后 48 小时内仍然受到抑制。CK 和 AST 的浓度在失败方案后更高，表明与非失败方案相比，肌肉损伤更大。这些结果支持非失败方案来减少外周性疲劳和肌肉损伤，而中枢性疲劳似乎不受组配置的影响。
