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Lack of Evidence of Angiotensin-Converting Enzyme 2 Expression and Replicative Infection by SARS-CoV-2 in Human Endothelial Cells.缺乏血管紧张素转换酶2在人内皮细胞中的表达及SARS-CoV-2复制性感染的证据。
Circulation. 2021 Feb 23;143(8):865-868. doi: 10.1161/CIRCULATIONAHA.120.052824. Epub 2021 Jan 6.
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Hypoxia alters the expression of ACE2 and TMPRSS2 SARS-CoV-2 cell entry mediators in hCMEC/D3 brain endothelial cells.缺氧改变 hCMEC/D3 脑内皮细胞中 ACE2 和 TMPRSS2 SARS-CoV-2 细胞进入介质的表达。
Microvasc Res. 2021 Nov;138:104232. doi: 10.1016/j.mvr.2021.104232. Epub 2021 Aug 18.
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Epstein-Barr Virus Lytic Replication Induces ACE2 Expression and Enhances SARS-CoV-2 Pseudotyped Virus Entry in Epithelial Cells.疱疹病毒 4 型（EBV）裂解复制诱导 ACE2 表达并增强 SARS-CoV-2 假型病毒进入上皮细胞。
J Virol. 2021 Jun 10;95(13):e0019221. doi: 10.1128/JVI.00192-21.
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SARS-CoV-2 Spike Protein Destabilizes Microvascular Homeostasis.SARS-CoV-2 刺突蛋白破坏微血管内稳态。
Microbiol Spectr. 2021 Dec 22;9(3):e0073521. doi: 10.1128/Spectrum.00735-21.
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Remodeling of intracellular architecture during SARS-CoV-2 infection of human endothelium.在 SARS-CoV-2 感染人类内皮细胞期间细胞内结构的重塑。
Sci Rep. 2024 Nov 30;14(1):29784. doi: 10.1038/s41598-024-80351-z.
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Multisystemic Cellular Tropism of SARS-CoV-2 in Autopsies of COVID-19 Patients.COVID-19 患者尸检中 SARS-CoV-2 的多系统细胞嗜性。
Cells. 2021 Jul 27;10(8):1900. doi: 10.3390/cells10081900.
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Extracellular vimentin is an attachment factor that facilitates SARS-CoV-2 entry into human endothelial cells.细胞外中间丝相关蛋白是一种附着因子，可促进 SARS-CoV-2 进入人内皮细胞。
Proc Natl Acad Sci U S A. 2022 Feb 8;119(6). doi: 10.1073/pnas.2113874119.
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SARS-COV2 Alters Blood Brain Barrier Integrity Contributing to Neuro-Inflammation.严重急性呼吸综合征冠状病毒2（SARS-CoV-2）改变血脑屏障完整性，导致神经炎症。
J Neuroimmune Pharmacol. 2021 Mar;16(1):4-6. doi: 10.1007/s11481-020-09975-y. Epub 2021 Jan 6.
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Single-cell transcriptional profile of ACE2 in healthy and failing human hearts.健康和衰竭人类心脏中ACE2的单细胞转录谱
Sci China Life Sci. 2021 Apr;64(4):652-655. doi: 10.1007/s11427-020-1787-5. Epub 2020 Sep 1.
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SARS-CoV-2 Entry Receptor ACE2 Is Expressed on Very Small CD45 Precursors of Hematopoietic and Endothelial Cells and in Response to Virus Spike Protein Activates the Nlrp3 Inflammasome.SARS-CoV-2 进入受体 ACE2 表达于造血和内皮细胞的非常小的 CD45 前体上，并且响应病毒刺突蛋白激活 Nlrp3 炎性小体。
Stem Cell Rev Rep. 2021 Feb;17(1):266-277. doi: 10.1007/s12015-020-10010-z.
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1
Selective Inhibition of Vascular Smooth Muscle Cell Function by COVID-19 Antiviral Drugs: Impact of Heme Oxygenase-1.新冠抗病毒药物对血管平滑肌细胞功能的选择性抑制：血红素加氧酶-1的影响
Antioxidants (Basel). 2025 Jul 31;14(8):945. doi: 10.3390/antiox14080945.
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Vasoprotective effects of lysophosphatidic acid inhibit vascular injury caused by SARS-CoV-2 infection.溶血磷脂酸的血管保护作用可抑制由SARS-CoV-2感染引起的血管损伤。
Sci Rep. 2025 Jul 25;15(1):24622. doi: 10.1038/s41598-025-06569-7.
3
SARS-CoV-2 infection promotes lung thrombosis by inducing integrinβ3 expression in vascular endothelial cells.严重急性呼吸综合征冠状病毒2（SARS-CoV-2）感染通过诱导血管内皮细胞中的整合素β3表达来促进肺血栓形成。
Sci Rep. 2025 Jul 1;15(1):20447. doi: 10.1038/s41598-025-02784-4.
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CXCL10-dependent epithelial-vascular cross-talk for endothelial activation following SARS-CoV-2 infection.SARS-CoV-2感染后CXCL10依赖性上皮-血管相互作用促进内皮细胞活化
Sci Rep. 2025 Jul 1;15(1):21129. doi: 10.1038/s41598-025-08329-z.
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sST2 is a key outcome biomarker in COVID-19: insights from discovery randomized trial.可溶性生长刺激表达基因2蛋白（sST2）是新冠病毒病的关键预后生物标志物：来自探索性随机试验的见解
Sci Rep. 2025 Apr 24;15(1):14348. doi: 10.1038/s41598-025-95122-7.
6
CircSARS-CV2-N1368 from SARS-CoV-2 impairs endothelial cell function through the upregulation of ATF7 to activate TLR4/NF-κB/ROS signaling.来自严重急性呼吸综合征冠状病毒2（SARS-CoV-2）的环状RNA SARS-CV2-N1368通过上调激活转录因子7（ATF7）来激活Toll样受体4（TLR4）/核因子κB（NF-κB）/活性氧（ROS）信号通路，从而损害内皮细胞功能。
Acta Pharmacol Sin. 2025 Mar 11. doi: 10.1038/s41401-025-01516-8.
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Challenging the notion of endothelial infection by SARS-CoV-2: insights from the current scientific evidence.挑战新冠病毒感染内皮细胞的观念：基于当前科学证据的见解
Front Immunol. 2025 Feb 4;16:1443932. doi: 10.3389/fimmu.2025.1443932. eCollection 2025.
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Innate immune sensors and regulators at the blood brain barrier: focus on toll-like receptors and inflammasomes as mediators of neuro-immune crosstalk and inflammation.血脑屏障处的天然免疫传感器与调节因子：聚焦于作为神经免疫串扰和炎症介质的Toll样受体和炎性小体。
J Neuroinflammation. 2025 Feb 15;22(1):39. doi: 10.1186/s12974-025-03360-3.
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SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells.严重急性呼吸综合征冠状病毒2（SARS-CoV-2）刺突蛋白（S蛋白）的表达促使内皮细胞形成多核巨细胞。
Sci Rep. 2025 Jan 28;15(1):3549. doi: 10.1038/s41598-025-86242-1.
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Sustained Vascular Inflammatory Effects of SARS-CoV-2 Spike Protein on Human Endothelial Cells.严重急性呼吸综合征冠状病毒2（SARS-CoV-2）刺突蛋白对人内皮细胞的持续血管炎症作用。
Inflammation. 2024 Dec 31. doi: 10.1007/s10753-024-02208-x.
本文引用的文献
1
Cells of the adult human heart.成人心脏细胞。
Nature. 2020 Dec;588(7838):466-472. doi: 10.1038/s41586-020-2797-4. Epub 2020 Sep 24.
2
Endotheliopathy Is Induced by Plasma From Critically Ill Patients and Associated With Organ Failure in Severe COVID-19.内皮病变由危重症患者的血浆诱导产生，并与重症 COVID-19 中的器官衰竭相关。
Circulation. 2020 Nov 10;142(19):1881-1884. doi: 10.1161/CIRCULATIONAHA.120.050907. Epub 2020 Sep 24.
3
Endothelial dysfunction in COVID-19: a position paper of the ESC Working Group for Atherosclerosis and Vascular Biology, and the ESC Council of Basic Cardiovascular Science.新型冠状病毒肺炎中的血管内皮功能障碍：欧洲心脏病学会动脉粥样硬化和血管生物学工作组以及欧洲心脏病学会心血管基础科学理事会立场文件。
Cardiovasc Res. 2020 Dec 1;116(14):2177-2184. doi: 10.1093/cvr/cvaa230.
4
Dendritic cell-specific antigen delivery by coronavirus vaccine vectors induces long-lasting protective antiviral and antitumor immunity.冠状病毒疫苗载体介导的树突状细胞特异性抗原递呈诱导持久的保护性抗病毒和抗肿瘤免疫。
mBio. 2010 Sep 14;1(4):e00171-10. doi: 10.1128/mBio.00171-10.
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