• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

[勘误] KLF4 和 ZEB1 之间的新型串扰调节胰腺导管腺癌对吉西他滨的耐药性。

[Corrigendum] Novel crosstalk between KLF4 and ZEB1 regulates gemcitabine resistance in pancreatic ductal adenocarcinoma.

机构信息

Department of Hepatobiliary Surgery, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong 250021, P.R. China.

Department of Paediatrics, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong 250021, P.R. China.

出版信息

Int J Oncol. 2021 Feb;58(2):278-279. doi: 10.3892/ijo.2020.5157. Epub 2020 Dec 10.

DOI:10.3892/ijo.2020.5157
PMID:33491746
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7864007/
Abstract

Subsequently to the publication of the above article, an interested reader drew to the attention of the Editorial Office that, in Fig. 1C on p. 1242, the flow cytometric images contained what appeared to be regular and repeating groups of cells. The office consequently asked the authors to provide the raw data for these images, as they would have been generated from the printouts, and the authors were able to demonstrate that these apparent anomalies were not contained in the original data. It is possible that the anomalous appearance of the data in this Figure may have resulted either from low resolution of the images, or the Figure itself may have been compressed. We are reprinting Fig. 1C opposite, highlighting the data of interest in greater detail. We trust that this satisfies the concerns of the reader in this instance, and thank them for their enquiry to the Editorial Office. The authors also requested that, after having provided the raw data of the original image in order to clarify the concerns of the reader, they may republish Fig. 1 featuring alternative data for Fig. 1C. The revised version of Fig. 1 is consequently shown on the next page. In this figure, flow cytometric analysis demonstrated that treatment with 10 µM gemcitabine induced the death of 66.5% of the BxPC‑3 cells, 29.54% of the Panc‑1 cells, and 34.52% of the MIApaca‑2 cells (Fig. 1C). The authors confirm that these data support the main conclusions presented in their paper, and are grateful to the Editor of International Journal of Oncology for allowing them this opportunity to publish a Corrigendum. They also apologise to the readership for any inconvenience caused. [the original article was published in International Journal of Oncology 51: 1239‑1248, 2017; DOI: 10.3892/ijo.2017.4099].

摘要

在上述文章发表后,一位感兴趣的读者向编辑部指出,在第 1242 页的图 1C 中,流式细胞术图像中似乎包含有规则且重复的细胞群。编辑部因此要求作者提供这些图像的原始数据,因为这些数据是从打印输出中生成的,而作者能够证明这些看似异常的数据并不包含在原始数据中。图 1 中数据的异常外观可能是由于图像分辨率低,或者图像本身被压缩。我们重新印刷了图 1C,突出显示了更详细的感兴趣的数据。我们相信这满足了读者在这种情况下的关注,并感谢他们向编辑部提出的询问。作者还要求,在提供原始图像的原始数据以澄清读者的担忧之后,他们可以重新发表图 1,并为图 1C 提供替代数据。因此,下一页显示了修订后的图 1。在该图中,流式细胞术分析表明,用 10µM 吉西他滨处理诱导 66.5%的 BxPC-3 细胞、29.54%的 Panc-1 细胞和 34.52%的 MIApaca-2 细胞死亡(图 1C)。作者确认这些数据支持他们在论文中提出的主要结论,并感谢国际肿瘤学杂志的编辑允许他们有机会发表更正。他们还向读者表示歉意,给他们带来了任何不便。[原始文章发表于国际肿瘤学杂志 51:1239-1248, 2017;DOI:10.3892/ijo.2017.4099]。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b4d2/7864007/a801eb1b56ab/IJO-58-02-0278-g01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b4d2/7864007/ef3cf2d7b7af/IJO-58-02-0278-g00.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b4d2/7864007/a801eb1b56ab/IJO-58-02-0278-g01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b4d2/7864007/ef3cf2d7b7af/IJO-58-02-0278-g00.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b4d2/7864007/a801eb1b56ab/IJO-58-02-0278-g01.jpg

相似文献

1
[Corrigendum] Novel crosstalk between KLF4 and ZEB1 regulates gemcitabine resistance in pancreatic ductal adenocarcinoma.[勘误] KLF4 和 ZEB1 之间的新型串扰调节胰腺导管腺癌对吉西他滨的耐药性。
Int J Oncol. 2021 Feb;58(2):278-279. doi: 10.3892/ijo.2020.5157. Epub 2020 Dec 10.
2
[Corrigendum] Mutation of TGF‑β receptor II facilitates human bladder cancer progression through altered TGF‑β1 signaling pathway.[勘误] TGF-β 受体 II 突变通过改变 TGF-β1 信号通路促进人膀胱癌的进展。
Int J Oncol. 2021 Feb;58(2):280. doi: 10.3892/ijo.2020.5161. Epub 2020 Dec 14.
3
[Corrigendum] Macrophage‑secreted IL‑8 induces epithelial‑mesen-chymal transition in hepatocellular carcinoma cells by activating the JAK2/STAT3/Snail pathway.[勘误] 巨噬细胞分泌的白细胞介素-8通过激活JAK2/STAT3/蜗牛蛋白通路诱导肝癌细胞发生上皮-间质转化。
Int J Oncol. 2024 Sep;65(3). doi: 10.3892/ijo.2024.5672. Epub 2024 Jul 26.
4
[Corrigendum] Repression of Smad4 by miR‑205 moderates TGF‑β-induced epithelial‑mesenchymal transition in A549 cell lines.[勘误] miR-205 对 Smad4 的抑制作用调节了 A549 细胞系中 TGF-β诱导的上皮-间充质转化。
Int J Oncol. 2021 Feb;58(2):276-277. doi: 10.3892/ijo.2021.5166. Epub 2021 Jan 4.
5
[Corrigendum] SB‑T‑121205, a next‑generation taxane, enhances apoptosis and inhibits migration/invasion in MCF‑7/PTX cells.[勘误] SB-T-121205,一种新一代紫杉烷,增强MCF-7/PTX细胞的凋亡并抑制其迁移/侵袭。
Int J Oncol. 2023 Jan;62(1). doi: 10.3892/ijo.2022.5463. Epub 2022 Dec 9.
6
[Corrigendum] High PLK4 expression promotes tumor progression and induces epithelial‑mesenchymal transition by regulating the Wnt/β‑catenin signaling pathway in colorectal cancer.[勘误] 高PLK4表达通过调节结直肠癌中的Wnt/β-连环蛋白信号通路促进肿瘤进展并诱导上皮-间质转化。
Int J Oncol. 2021 Jun;58(6). doi: 10.3892/ijo.2021.5213. Epub 2021 May 6.
7
[Corrigendum] Role of the EZH2/miR‑200 axis in STAT3‑mediated OSCC invasion.[勘误] EZH2/miR-200轴在STAT3介导的口腔鳞状细胞癌侵袭中的作用
Int J Oncol. 2023 Jul;63(1). doi: 10.3892/ijo.2023.5528. Epub 2023 Jun 2.
8
[Corrigendum] Dexamethasone-induced inhibition of miR-132 via methylation promotes TGF-β-driven progression of pancreatic cancer.[勘误] 地塞米松通过甲基化诱导的miR-132抑制促进TGF-β驱动的胰腺癌进展。
Int J Oncol. 2023 Aug;63(2). doi: 10.3892/ijo.2023.5543. Epub 2023 Jul 7.
9
[Corrigendum] S100A9 promotes the proliferation and invasion of HepG2 hepatocellular carcinoma cells via the activation of the MAPK signaling pathway.[勘误] S100A9通过激活丝裂原活化蛋白激酶(MAPK)信号通路促进HepG2肝癌细胞的增殖和侵袭。
Int J Oncol. 2023 Apr;62(4). doi: 10.3892/ijo.2023.5494. Epub 2023 Mar 3.
10
[Corrigendum] Epithelial mesenchymal transition induced by the CXCL9/CXCR3 axis through AKT activation promotes invasion and metastasis in tongue squamous cell carcinoma.[勘误] CXCL9/CXCR3 轴通过 AKT 激活诱导的上皮间质转化促进舌鳞癌细胞侵袭和转移。
Oncol Rep. 2021 Feb;45(2):791-792. doi: 10.3892/or.2020.7883. Epub 2020 Dec 3.

引用本文的文献

1
KLF4 transcription factor in tumorigenesis.肿瘤发生中的KLF4转录因子。
Cell Death Discov. 2023 Apr 8;9(1):118. doi: 10.1038/s41420-023-01416-y.