Girard J P, Fernandes B
Schweiz Med Wochenschr. 1977 Oct 15;107(41):1462.
This study demonstrates that, in spite of the fairly normal functional capacity of T lymphocytes in sarcoidosis, a deficit of cell-mediated immunity frequently develops. It is shown that monocytes of sarcoidosis seem to share in responsibility for the appearance of this deficit. Soluble factors from sensitized healthy monocytes are shown to be capable of restoring this capacity.
本研究表明,尽管结节病中T淋巴细胞的功能能力相当正常,但细胞介导的免疫缺陷仍经常出现。研究表明,结节病的单核细胞似乎对这种缺陷的出现负有责任。已证明来自致敏健康单核细胞的可溶性因子能够恢复这种能力。