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Correction: G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer's disease pathology.更正:G82S RAGE多态性影响与阿尔茨海默病病理学相关的淀粉样蛋白-RAGE相互作用。
PLoS One. 2021 Mar 4;16(3):e0248252. doi: 10.1371/journal.pone.0248252. eCollection 2021.
2
G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer's disease pathology.G82S RAGE 多态性影响阿尔茨海默病病理相关的淀粉样蛋白-RAGE 相互作用。
PLoS One. 2020 Oct 29;15(10):e0225487. doi: 10.1371/journal.pone.0225487. eCollection 2020.
3
The association between the RAGE G82S polymorphism, sRAGE and chronic periodontitis in Taiwanese individuals with and without diabetes.台湾糖尿病患者与非糖尿病患者中晚期糖基化终末产物受体(RAGE)G82S多态性、可溶性RAGE(sRAGE)与慢性牙周炎之间的关联。
J Periodontal Res. 2015 Dec;50(6):881-9. doi: 10.1111/jre.12282. Epub 2015 May 18.
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G82S polymorphism of receptor for advanced glycation end products gene and serum soluble RAGE levels in mild cognitive impairment and dementia of Alzheimer's type patients in Turkish population.土耳其人群中晚期糖基化终末产物受体基因的G82S多态性与轻度认知障碍及阿尔茨海默病型痴呆患者血清可溶性RAGE水平的关系
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De-N-glycosylation or G82S mutation of RAGE sensitizes its interaction with advanced glycation endproducts.RAGE的去N-糖基化或G82S突变增强了其与晚期糖基化终产物的相互作用。
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Correction: Quantitative Amyloid Imaging in Autosomal Dominant Alzheimer's Disease: Results from the DIAN Study Group.更正:常染色体显性阿尔茨海默病的定量淀粉样蛋白成像:DIAN研究组的结果。
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Association of the Gly82Ser polymorphism in the receptor for advanced glycation end products (RAGE) gene with circulating levels of soluble RAGE and inflammatory markers in nondiabetic and nonobese Koreans.晚期糖基化终末产物受体(RAGE)基因中Gly82Ser多态性与非糖尿病和非肥胖韩国人可溶性RAGE循环水平及炎症标志物的关联。
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APOE ε4 Carriers Have a Greater Propensity to Glycation and sRAGE Which Is Further Influenced by RAGE G82S Polymorphism.载脂蛋白 E ε4 携带者更容易发生糖基化和 sRAGE,而 RAGE G82S 多态性进一步影响了这一过程。
J Gerontol A Biol Sci Med Sci. 2020 Sep 25;75(10):1899-1905. doi: 10.1093/gerona/glz259.
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Correction: Chronic Anatabine Treatment Reduces Alzheimer's Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of AD.更正:慢性新烟草碱治疗可减轻阿尔茨海默病(AD)样病理,并改善AD转基因小鼠模型中的社会行为缺陷。
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Post-translational modifications of beta-amyloid alter its transport in the blood-brain barrier model.β-淀粉样蛋白的翻译后修饰改变其在血脑屏障模型中的转运。
Front Mol Neurosci. 2024 Mar 7;17:1362581. doi: 10.3389/fnmol.2024.1362581. eCollection 2024.
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Effect of β-amyloid on blood-brain barrier properties and function.β-淀粉样蛋白对血脑屏障特性及功能的影响。
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Docking and Molecular Dynamics-Based Identification of Interaction between Various Beta-Amyloid Isoforms and RAGE Receptor.基于对接和分子动力学的不同β-淀粉样蛋白异构体与 RAGE 受体相互作用的鉴定。
Int J Mol Sci. 2022 Oct 5;23(19):11816. doi: 10.3390/ijms231911816.
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Research Progress on the Role of Microglia Membrane Proteins or Receptors in Neuroinflammation and Degeneration.小胶质细胞膜蛋白或受体在神经炎症和神经退行性变中的作用研究进展
Front Cell Neurosci. 2022 Feb 25;16:831977. doi: 10.3389/fncel.2022.831977. eCollection 2022.

本文引用的文献

1
G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer's disease pathology.G82S RAGE 多态性影响阿尔茨海默病病理相关的淀粉样蛋白-RAGE 相互作用。
PLoS One. 2020 Oct 29;15(10):e0225487. doi: 10.1371/journal.pone.0225487. eCollection 2020.

更正:G82S RAGE多态性影响与阿尔茨海默病病理学相关的淀粉样蛋白-RAGE相互作用。

Correction: G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer's disease pathology.

作者信息

Chellappa Rani Cathrine, Lukose Bincy, Rani P

出版信息

PLoS One. 2021 Mar 4;16(3):e0248252. doi: 10.1371/journal.pone.0248252. eCollection 2021.

DOI:10.1371/journal.pone.0248252
PMID:33662008
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7932165/
Abstract

[This corrects the article DOI: 10.1371/journal.pone.0225487.].

摘要

[本文更正文章DOI:10.1371/journal.pone.0225487。]