聚团肠杆菌脂多糖诱导的肺表面活性物质变化作为棉尘病发病机制中的一个因素。
Enterobacter agglomerans lipopolysaccharide-induced changes in pulmonary surfactant as a factor in the pathogenesis of byssinosis.
作者信息
DeLucca A J, Brogden K A, Engen R
机构信息
Composition and Properties Research Unit, Southern Regional Research Center, New Orleans, Louisiana 70124.
出版信息
J Clin Microbiol. 1988 Apr;26(4):778-80. doi: 10.1128/jcm.26.4.778-780.1988.
Abstract
Lipopolysaccharide (LPS) from Enterobacter agglomerans and pulmonary surfactant mixtures were centrifuged in discontinuous sucrose gradients to determine whether LPS bound to surfactant and examined in a Langmuir trough with a Wilhelmy balance to determine whether LPS altered the surface activity of surfactant. The LPS was found to bind to the surfactant and altered its surface tension properties. The binding of LPS to surfactant in the lung may change the physiological properties of surfactant and be a possible mechanism for the pathogenesis of byssinosis.
摘要
将来自成团肠杆菌的脂多糖（LPS）与肺表面活性剂混合物在不连续蔗糖梯度中进行离心，以确定LPS是否与表面活性剂结合，并在带有威尔海姆天平的朗缪尔槽中进行检测，以确定LPS是否改变了表面活性剂的表面活性。结果发现LPS与表面活性剂结合并改变了其表面张力特性。LPS在肺中与表面活性剂的结合可能会改变表面活性剂的生理特性，并且可能是棉尘病发病机制的一种可能机制。
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