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Emerging role of RNA m6A modification in chronic pain.

作者信息

Albik Sfian, Tao Yuan-Xiang

机构信息

Departments of Anesthesiology.

Physiology, Pharmacology, and Neuroscience, and.

出版信息

Pain. 2021 Jul 1;162(7):1897-1898. doi: 10.1097/j.pain.0000000000002219.

DOI:10.1097/j.pain.0000000000002219
PMID:33675633
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8205953/
Abstract
摘要

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本文引用的文献

1
Methyltransferase-like 3 contributes to inflammatory pain by targeting TET1 in YTHDF2-dependent manner.甲基转移酶样蛋白 3 通过 YTHDF2 依赖性靶向 TET1 促进炎性疼痛。
Pain. 2021 Jul 1;162(7):1960-1976. doi: 10.1097/j.pain.0000000000002218.
2
N-Methyladenosine Demethylase FTO Contributes to Neuropathic Pain by Stabilizing G9a Expression in Primary Sensory Neurons.N-甲基腺苷去甲基化酶FTO通过稳定初级感觉神经元中的G9a表达促进神经性疼痛。
Adv Sci (Weinh). 2020 May 27;7(13):1902402. doi: 10.1002/advs.201902402. eCollection 2020 Jul.
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RNA-seq of spinal cord from nerve-injured rats after spinal cord stimulation.脊髓刺激后神经损伤大鼠脊髓的 RNA-seq 分析。
Mol Pain. 2018 Jan-Dec;14:1744806918817429. doi: 10.1177/1744806918817429. Epub 2018 Nov 19.
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Long noncoding RNA (lncRNA): a target in neuropathic pain.长链非编码 RNA(lncRNA):神经病理性疼痛的靶点。
Expert Opin Ther Targets. 2019 Jan;23(1):15-20. doi: 10.1080/14728222.2019.1550075. Epub 2018 Dec 2.
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The Role of mA/m-RNA Methylation in Stress Response Regulation.mA/m-RNA 甲基化在应激反应调控中的作用。
Neuron. 2018 Jul 25;99(2):389-403.e9. doi: 10.1016/j.neuron.2018.07.009.
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Dynamic transcriptomic mA decoration: writers, erasers, readers and functions in RNA metabolism.动态转录组 mA 修饰:RNA 代谢中的写入器、擦除器、读取器及其功能。
Cell Res. 2018 Jun;28(6):616-624. doi: 10.1038/s41422-018-0040-8. Epub 2018 May 22.
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The Emerging Field of Epitranscriptomics in Neurodevelopmental and Neuronal Disorders.表观转录组学在神经发育和神经元疾病中的新兴领域
Front Bioeng Biotechnol. 2018 Apr 13;6:46. doi: 10.3389/fbioe.2018.00046. eCollection 2018.
8
DNA Hydroxymethylation by Ten-eleven Translocation Methylcytosine Dioxygenase 1 and 3 Regulates Nociceptive Sensitization in a Chronic Inflammatory Pain Model.由11-易位甲基胞嘧啶双加氧酶1和3介导的DNA羟甲基化在慢性炎症性疼痛模型中调节伤害性敏感化。
Anesthesiology. 2017 Jul;127(1):147-163. doi: 10.1097/ALN.0000000000001632.
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G9a inhibits CREB-triggered expression of mu opioid receptor in primary sensory neurons following peripheral nerve injury.G9a抑制外周神经损伤后初级感觉神经元中CREB触发的μ阿片受体表达。
Mol Pain. 2016 Dec 7;12. doi: 10.1177/1744806916682242. Print 2016.
10
G9a participates in nerve injury-induced Kcna2 downregulation in primary sensory neurons.G9a 参与初级感觉神经元神经损伤诱导的 Kcna2 下调。
Sci Rep. 2016 Nov 22;6:37704. doi: 10.1038/srep37704.