Tight Junction Proteins Join the Local Force for Bulk Endocytosis and Microvillus Inclusion.
作者信息
Bandyopadhyay Sheila, Bonder Edward, Gao Nan
机构信息
Department of Biological Sciences, Rutgers University, Newark, New Jersey.
Department of Biological Sciences, Rutgers University, Newark, New Jersey.
出版信息
Cell Mol Gastroenterol Hepatol. 2021;12(1):348-349. doi: 10.1016/j.jcmgh.2021.02.012. Epub 2021 Mar 20.
Abstract
摘要
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本文引用的文献
1
Recruitment of Polarity Complexes and Tight Junction Proteins to the Site of Apical Bulk Endocytosis.极性复合物和紧密连接蛋白在顶端 bulk 内吞位点的募集。
Cell Mol Gastroenterol Hepatol. 2021;12(1):59-80. doi: 10.1016/j.jcmgh.2021.01.022. Epub 2021 Feb 3.
2
Loss of myosin Vb promotes apical bulk endocytosis in neonatal enterocytes.肌球蛋白 Vb 的缺失促进了新生肠细胞的顶端 bulk 胞吞作用。
J Cell Biol. 2019 Nov 4;218(11):3647-3662. doi: 10.1083/jcb.201902063. Epub 2019 Sep 27.
3
Intestinal epithelial cell polarity defects in disease: lessons from microvillus inclusion disease.疾病中肠上皮细胞极性缺陷：微绒毛包涵病的启示。
Dis Model Mech. 2018 Feb 13;11(2):dmm031088. doi: 10.1242/dmm.031088.
4
Disruption of Rab8a and Rab11a causes formation of basolateral microvilli in neonatal enteropathy.Rab8a和Rab11a的破坏会导致新生儿肠病中基底外侧微绒毛的形成。
J Cell Sci. 2017 Aug 1;130(15):2491-2505. doi: 10.1242/jcs.201897. Epub 2017 Jun 8.
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Mechanisms of Cell Polarity-Controlled Epithelial Homeostasis and Immunity in the Intestine.肠道中细胞极性控制的上皮稳态和免疫机制
Cold Spring Harb Perspect Biol. 2017 Jul 5;9(7):a027888. doi: 10.1101/cshperspect.a027888.
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The localisation of the apical Par/Cdc42 polarity module is specifically affected in microvillus inclusion disease.顶端Par/Cdc42极性模块的定位在微绒毛包涵体病中受到特异性影响。
Biol Cell. 2016 Jan;108(1):19-28. doi: 10.1111/boc.201500034. Epub 2015 Dec 8.
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Microvillus inclusion disease: loss of Myosin vb disrupts intracellular traffic and cell polarity.微绒毛包涵体病：Myosin vb 的缺失破坏细胞内运输和细胞极性。
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Curr Biol. 2012 Jun 19;22(12):1116-22. doi: 10.1016/j.cub.2012.04.020. Epub 2012 May 31.
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