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伊朗职业足球运动员的控制缺陷、影响因素与带伤带痛比赛。

Control Deficits, Conditioning Factors, and Playing through Pain and Injury among Iranian Professional Soccer Players.

机构信息

Department of Humanities and Social Sciences, University of Mazandaran, Babolsar 4741613534, Iran.

Department of Security Studies and Criminal Justice, Angelo State University, San Angelo, TX 76901, USA.

出版信息

Int J Environ Res Public Health. 2021 Mar 25;18(7):3387. doi: 10.3390/ijerph18073387.

DOI:10.3390/ijerph18073387
PMID:33805863
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8036426/
Abstract

Playing through pain and injury is a common and accepted behavior in the athletic realm. The purpose of this research was to apply Tittle's control balance theory to explain why athletes engage in playing through pain and injury despite its risky nature. We hypothesized that playing through pain and injury is a form of submission described by Tittle and that it can be predicted by the concept of control deficit. To this end, we collected and used data from a sample of 410 professional soccer players from Guilan province, Iran, and tested several propositions derived from control balance theory. Hierarchical linear regression was used to analyze the data. The study findings demonstrate that players with more control deficits are more likely to play through pain and injury. This relationship is conditioned by self-control, opportunity, motivation, perceived benefits, and provocations. For example, the relationship between control deficit and playing through pain and injury is stronger for those with lower self-control. Our findings support the utility of control balance theory in explaining an act of submission (i.e., playing through pain and injury).

摘要

在竞技领域,带伤参赛是一种常见且被广泛接受的行为。本研究旨在运用蒂特的控制平衡理论来解释为什么运动员尽管知道其风险,仍会带伤参赛。我们假设带伤参赛是蒂特所描述的一种顺从行为,并且可以通过控制缺陷的概念来预测。为此,我们从伊朗吉兰省的 410 名职业足球运动员中收集并使用了数据,并检验了控制平衡理论得出的几个命题。我们采用分层线性回归来分析数据。研究结果表明,控制缺陷较大的运动员更有可能带伤参赛。这种关系受到自我控制、机会、动机、感知收益和挑衅的制约。例如,对于自我控制较低的运动员来说,控制缺陷与带伤参赛之间的关系更强。我们的研究结果支持了控制平衡理论在解释顺从行为(即带伤参赛)方面的效用。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/e638e50fd2a3/ijerph-18-03387-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/0f9838bb07c8/ijerph-18-03387-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/39b737ce69bb/ijerph-18-03387-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/efa1a74783a2/ijerph-18-03387-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/58ef24fb7c44/ijerph-18-03387-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/199b7f9fb665/ijerph-18-03387-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/dbe84d334630/ijerph-18-03387-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/e638e50fd2a3/ijerph-18-03387-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/0f9838bb07c8/ijerph-18-03387-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/39b737ce69bb/ijerph-18-03387-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/efa1a74783a2/ijerph-18-03387-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/58ef24fb7c44/ijerph-18-03387-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/199b7f9fb665/ijerph-18-03387-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/dbe84d334630/ijerph-18-03387-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c8a/8036426/e638e50fd2a3/ijerph-18-03387-g007.jpg

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