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13q12.2 deletions and FLT3 overexpression in acute leukemias.

作者信息

Poubel Caroline Pires, Boroni Mariana, Emerenciano Mariana

机构信息

Division of Clinical Research and.

Bioinformatics and Computational Biology Laboratory, Research Center, Instituto Nacional de Câncer (INCA), Rio de Janeiro, Brazil.

出版信息

Blood Adv. 2021 Apr 27;5(8):2075-2078. doi: 10.1182/bloodadvances.2020003643.

Abstract
摘要

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本文引用的文献

1
Deletions in BCP-ALL result in a TAD more FLT3.
Blood. 2020 Aug 20;136(8):919-920. doi: 10.1182/blood.2020006607.
3
FLT3 overexpression in acute leukaemias: New insights into the search for molecular mechanisms.
Biochim Biophys Acta Rev Cancer. 2019 Aug;1872(1):80-88. doi: 10.1016/j.bbcan.2019.06.001. Epub 2019 Jun 12.
4
5
FLT3 pathway is a potential therapeutic target for PRC2-mutated T-cell acute lymphoblastic leukemia.
Blood. 2018 Dec 6;132(23):2520-2524. doi: 10.1182/blood-2018-04-845628. Epub 2018 Oct 3.
6
High expression of FLT3 is a risk factor in leukemia.
Mol Med Rep. 2018 Feb;17(2):2885-2892. doi: 10.3892/mmr.2017.8232. Epub 2017 Dec 8.
8
RNA binding protein MSI2 positively regulates FLT3 expression in myeloid leukemia.
Leuk Res. 2017 Mar;54:47-54. doi: 10.1016/j.leukres.2017.01.015. Epub 2017 Jan 11.

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