Bahls F H, Chatrian G E, Mesher R A, Sumi S M, Ruff R L
Department of Medicine, University of Washington School of Medicine, Seattle.
Neurology. 1988 Sep;38(9):1490-3. doi: 10.1212/wnl.38.9.1490.
A 61-year-old man became deaf after the second of two cerebral infarctions which successively involved the temporal and adjacent cortices. He remained completely deaf until death 27 months later. Click stimulation demonstrated normal short-latency potentials, middle-latency responses better developed to stimulation of the right than of the left ear, and absent long-latency potentials. Neuropathologic examination showed cystic infarctions involving both transverse temporal gyri and adjacent cortical areas with preservation of the brainstem auditory nuclei. Persistent deafness can result from bilateral lesions involving the auditory and adjoining cortices.
一名61岁男性在先后累及颞叶及相邻皮质的两次脑梗死中的第二次发作后失聪。直至27个月后死亡，他一直完全失聪。点击刺激显示短潜伏期电位正常，中潜伏期反应在刺激右耳时比左耳时更明显，且长潜伏期电位缺失。神经病理学检查显示双侧颞横回及相邻皮质区域出现囊性梗死，脑干听觉核保存。双侧累及听觉及相邻皮质的病变可导致持续性耳聋。
