• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

相似文献

1
Reply to Eisenhut.回复艾森胡特。
Am J Physiol Lung Cell Mol Physiol. 2021 Jul 1;321(1):L287-L289. doi: 10.1152/ajplung.00246.2021.
2
Cl- transport by cystic fibrosis transmembrane conductance regulator (CFTR) contributes to the inhibition of epithelial Na+ channels (ENaCs) in Xenopus oocytes co-expressing CFTR and ENaC.在共表达囊性纤维化跨膜传导调节因子（CFTR）和上皮钠通道（ENaC）的非洲爪蟾卵母细胞中，CFTR介导的氯离子转运有助于抑制ENaC。
J Physiol. 1998 May 1;508 ( Pt 3)(Pt 3):825-36. doi: 10.1111/j.1469-7793.1998.825bp.x.
3
The cytosolic termini of the beta- and gamma-ENaC subunits are involved in the functional interactions between cystic fibrosis transmembrane conductance regulator and epithelial sodium channel.β-ENaC和γ-ENaC亚基的胞质末端参与囊性纤维化跨膜传导调节因子与上皮钠通道之间的功能相互作用。
J Biol Chem. 2000 Sep 8;275(36):27947-56. doi: 10.1074/jbc.M002848200.
4
The cystic fibrosis transmembrane conductance regulator (CFTR) inhibits ENaC through an increase in the intracellular Cl- concentration.囊性纤维化跨膜传导调节因子（CFTR）通过增加细胞内氯离子浓度来抑制上皮钠通道（ENaC）。
EMBO Rep. 2001 Nov;2(11):1047-51. doi: 10.1093/embo-reports/kve232. Epub 2001 Oct 17.
5
Cystic fibrosis transmembrane conductance regulator-dependent up-regulation of Kir1.1 (ROMK) renal K+ channels by the epithelial sodium channel.上皮钠通道对囊性纤维化跨膜传导调节因子依赖性的肾钾通道Kir1.1（ROMK）的上调作用
J Biol Chem. 2002 Jul 12;277(28):25377-84. doi: 10.1074/jbc.M201925200. Epub 2002 May 6.
6
The effect of ambroxol on chloride transport, CFTR and ENaC in cystic fibrosis airway epithelial cells.氨溴索对囊性纤维化气道上皮细胞氯转运、CFTR 和 ENaC 的影响。
Cell Biol Int. 2013 Nov;37(11):1149-56. doi: 10.1002/cbin.10146. Epub 2013 Jul 23.
7
Cystic fibrosis transmembrane conductance regulator inhibits epithelial Na+ channels carrying Liddle's syndrome mutations.囊性纤维化跨膜传导调节因子抑制携带利德尔综合征突变的上皮钠通道。
J Biol Chem. 1999 May 14;274(20):13894-9. doi: 10.1074/jbc.274.20.13894.
8
Cystic fibrosis transmembrane conductance regulator differentially regulates human and mouse epithelial sodium channels in Xenopus oocytes.囊性纤维化跨膜传导调节因子在非洲爪蟾卵母细胞中对人和小鼠上皮钠通道进行差异性调节。
J Biol Chem. 2004 May 28;279(22):23183-92. doi: 10.1074/jbc.M402373200. Epub 2004 Mar 26.
9
Control of epithelial Na+ conductance by the cystic fibrosis transmembrane conductance regulator.囊性纤维化跨膜传导调节因子对上皮细胞钠离子电导的调控
Pflugers Arch. 2000 Jun;440(2):193-201. doi: 10.1007/s004240000255.
10
Molecular proximity of cystic fibrosis transmembrane conductance regulator and epithelial sodium channel assessed by fluorescence resonance energy transfer.通过荧光共振能量转移评估囊性纤维化跨膜传导调节因子与上皮钠通道的分子接近度。
J Biol Chem. 2007 Dec 14;282(50):36481-8. doi: 10.1074/jbc.M708089200. Epub 2007 Oct 3.

引用本文的文献

1
Competitive cleavage of SARS-CoV-2 spike protein and epithelial sodium channel by plasmin as a potential mechanism for COVID-19 infection.纤溶酶对 SARS-CoV-2 刺突蛋白和上皮钠离子通道的竞争裂解作用可能是 COVID-19 感染的机制之一。
Am J Physiol Lung Cell Mol Physiol. 2022 Nov 1;323(5):L569-L577. doi: 10.1152/ajplung.00152.2022. Epub 2022 Oct 4.

本文引用的文献

1
The significance of a lack of rhinorrhea in severe coronavirus 19 lung disease.严重新型冠状病毒19型肺病中无鼻漏的意义。
Am J Physiol Lung Cell Mol Physiol. 2021 Jun 1;320(6):L1194-L1195. doi: 10.1152/ajplung.00066.2021.
2
Cystic fibrosis improves COVID-19 survival and provides clues for treatment of SARS-CoV-2.囊性纤维化改善 COVID-19 患者存活率，并为治疗 SARS-CoV-2 提供线索。
Purinergic Signal. 2021 Sep;17(3):399-410. doi: 10.1007/s11302-021-09771-0. Epub 2021 May 10.
3
Dysregulation of ion transport in the lung epithelium infected with SARS-CoV-2.感染新冠病毒的肺上皮细胞中离子转运的失调。
Am J Physiol Lung Cell Mol Physiol. 2021 Jun 1;320(6):L1183-L1185. doi: 10.1152/ajplung.00170.2021. Epub 2021 Apr 21.
4
Molecular mechanisms of Na,K-ATPase dysregulation driving alveolar epithelial barrier failure in severe COVID-19.钠钾ATP酶失调驱动重症新型冠状病毒肺炎肺泡上皮屏障功能障碍的分子机制
Am J Physiol Lung Cell Mol Physiol. 2021 Jun 1;320(6):L1186-L1193. doi: 10.1152/ajplung.00056.2021. Epub 2021 Mar 9.
5
SARS-CoV-2 may hijack GPCR signaling pathways to dysregulate lung ion and fluid transport.SARS-CoV-2 可能劫持 GPCR 信号通路，使肺离子和液体转运失调。
Am J Physiol Lung Cell Mol Physiol. 2021 Mar 1;320(3):L430-L435. doi: 10.1152/ajplung.00499.2020. Epub 2021 Jan 12.
6
A Pathophysiological Model for COVID-19: Critical Importance of Transepithelial Sodium Transport upon Airway Infection.COVID-19 的病理生理学模型：气道感染时跨上皮钠转运的重要性。
Function (Oxf). 2020;1(2):zqaa024. doi: 10.1093/function/zqaa024. Epub 2020 Oct 6.
7
The global impact of SARS-CoV-2 in 181 people with cystic fibrosis.181 名囊性纤维化患者中 SARS-CoV-2 的全球影响。
J Cyst Fibros. 2020 Nov;19(6):868-871. doi: 10.1016/j.jcf.2020.10.003. Epub 2020 Nov 4.
8
First Wave of COVID-19 in French Patients with Cystic Fibrosis.法国囊性纤维化患者中的第一波新冠疫情
J Clin Med. 2020 Nov 10;9(11):3624. doi: 10.3390/jcm9113624.
9
Objective evaluation of the nasal mucosal secretion in COVID-19 patients with anosmia.目的评估 COVID-19 患者伴嗅觉丧失的鼻黏膜分泌物。
Ir J Med Sci. 2021 Aug;190(3):889-891. doi: 10.1007/s11845-020-02405-1. Epub 2020 Oct 19.
10
Are cystic fibrosis mutation carriers a potentially highly vulnerable group to COVID-19?囊性纤维化基因突变携带者是 COVID-19 的潜在高脆弱人群吗？
J Cell Mol Med. 2020 Nov;24(22):13542-13545. doi: 10.1111/jcmm.15941. Epub 2020 Oct 3.

Reply to Eisenhut.

作者信息

Abdel Hameid Reem, Cormet-Boyaka Estelle, Kuebler Wolfgang M, Uddin Mohammed, Berdiev Bakhrom K

机构信息

Mohammed Bin Rashid University of Medicine and Health Sciences, Dubai, United Arab Emirates.

Department of Veterinary Biosciences, The Ohio State University, Columbus, Ohio.

出版信息

Am J Physiol Lung Cell Mol Physiol. 2021 Jul 1;321(1):L287-L289. doi: 10.1152/ajplung.00246.2021.

DOI:10.1152/ajplung.00246.2021
PMID:34233142
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8270517/
Abstract
摘要