再喂养后持续的葡萄糖输出导致甲状腺功能亢进症中的葡萄糖不耐受。
Continued glucose output after re-feeding contributes to glucose intolerance in hyperthyroidism.
作者信息
Holness M J, Sugden M C
机构信息
Department of Chemical Pathology, London Hospital Medical College, U.K.
出版信息
Biochem J. 1987 Nov 1;247(3):801-4. doi: 10.1042/bj2470801.
Abstract
The effects of hyperthyroidism to elicit glucose intolerance after glucose administration were decreased under conditions where hepatic glucose output was suppressed. It is concluded that continued hepatic glucose output contributes to abnormal glucose tolerance in hyperthyroidism.
摘要
在肝葡萄糖输出受到抑制的情况下，甲状腺功能亢进症在给予葡萄糖后引发葡萄糖不耐受的作用减弱。得出的结论是，持续的肝葡萄糖输出导致甲状腺功能亢进症患者出现异常的葡萄糖耐量。
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本文引用的文献
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Regulation of hepatic lipogenesis in starved and diabetic animals by thyroid hormone.甲状腺激素对饥饿和糖尿病动物肝脏脂肪生成的调节作用。
Biosci Rep. 1981 Oct;1(10):757-64. doi: 10.1007/BF01114797.
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Liver glycogen synthase in rats with a glycogen-storage disorder. The role of glycogen in the regulation of glycogen synthase.患有糖原贮积症大鼠的肝脏糖原合酶。糖原在糖原合酶调节中的作用。
Eur J Biochem. 1980;108(1):73-7. doi: 10.1111/j.1432-1033.1980.tb04697.x.
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