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Liver glycogen synthase in rats with a glycogen-storage disorder. The role of glycogen in the regulation of glycogen synthase.

作者信息

Watts C, Malthus R S

出版信息

Eur J Biochem. 1980;108(1):73-7. doi: 10.1111/j.1432-1033.1980.tb04697.x.

DOI:10.1111/j.1432-1033.1980.tb04697.x
PMID:6773765
Abstract
摘要

相似文献

1
Liver glycogen synthase in rats with a glycogen-storage disorder. The role of glycogen in the regulation of glycogen synthase.患有糖原贮积症大鼠的肝脏糖原合酶。糖原在糖原合酶调节中的作用。
Eur J Biochem. 1980;108(1):73-7. doi: 10.1111/j.1432-1033.1980.tb04697.x.
2
The role of glycogen synthase phosphatase in the glucocorticoid-induced deposition of glycogen in foetal rat liver.糖原合酶磷酸酶在糖皮质激素诱导的胎鼠肝脏糖原沉积中的作用。
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3
Effects of diabetes and glycogen on the distribution between soluble and particulate fractions of activities of enzymes involved in hepatic glycogen metabolism.糖尿病和糖原对参与肝糖原代谢的酶活性在可溶性和颗粒性组分之间分布的影响。
Life Sci. 1978 Sep 11;23(10):1083-91. doi: 10.1016/0024-3205(78)90670-7.
4
The activation of glycogen synthase in hepatocytes from rats with a glycogen storage disorder (gsd/gsd).糖原贮积症(gsd/gsd)大鼠肝细胞中糖原合酶的激活
FEBS Lett. 1982 Aug 2;144(2):231-4. doi: 10.1016/0014-5793(82)80644-3.
5
Glucose activation of liver glycogen synthase. Insulin-mediated restoration of glucose effect in diabetic rats is blocked by protein synthesis inhibitor.肝脏糖原合酶的葡萄糖激活作用。糖尿病大鼠中胰岛素介导的葡萄糖效应恢复被蛋白质合成抑制剂阻断。
Biochim Biophys Acta. 1979 Feb 19;583(1):36-46. doi: 10.1016/0304-4165(79)90307-6.
6
Primer activity of a low molecular weight glycogen from a patient with an unusual glycogen storage disease.一名患有罕见糖原贮积病患者的低分子量糖原的引物活性
Physiol Chem Phys. 1972;4(1):56-60.
7
Effects of dexamethasone administration on hepatic glycogen synthesis and accumulation in adrenalectomized fasted rats.地塞米松给药对肾上腺切除的禁食大鼠肝脏糖原合成及蓄积的影响。
Endocrinology. 1984 Aug;115(2):625-9. doi: 10.1210/endo-115-2-625.
8
Rat liver glycogen metabolism in the perinatal period.围产期大鼠肝脏糖原代谢
Biochim Biophys Acta. 1979 Oct 4;587(2):145-54. doi: 10.1016/0304-4165(79)90349-0.
9
Low molecular weight glycogen as a cause of generalized glycogen storage disease.低分子量糖原作为全身性糖原贮积病的一个病因
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10
Glycogen metabolism in neonatal liver of the rat.大鼠新生肝脏中的糖原代谢
Arch Biochem Biophys. 1986 Sep;249(2):605-10. doi: 10.1016/0003-9861(86)90039-1.

引用本文的文献

1
A whole-body model for glycogen regulation reveals a critical role for substrate cycling in maintaining blood glucose homeostasis.全身性糖原调节模型揭示了底物循环在维持血糖稳态中的关键作用。
PLoS Comput Biol. 2011 Dec;7(12):e1002272. doi: 10.1371/journal.pcbi.1002272. Epub 2011 Dec 1.
2
Alpha 1-Adrenergic stimulation of Ca2+ mobilization without phosphorylase activation in hepatocytes from phosphorylase b kinase-deficient gsd/gsd rats.在磷酸化酶b激酶缺陷的gsd/gsd大鼠的肝细胞中,α1-肾上腺素能刺激Ca2+动员而不激活磷酸化酶。
Biochem J. 1981 Aug 15;198(2):379-83. doi: 10.1042/bj1980379.
3
Dephosphorylation of glycogen synthase in rat heart extracts by E. coli alkaline phosphatase. Use of an exogenous phosphatase to study substrate-mediated regulation of dephosphorylation.
大肠杆菌碱性磷酸酶对大鼠心脏提取物中糖原合酶的去磷酸化作用。利用外源性磷酸酶研究底物介导的去磷酸化调节。
Mol Cell Biochem. 1982 May 14;44(3):149-59. doi: 10.1007/BF00238503.
4
Difference in glucose sensitivity of liver glycolysis and glycogen synthesis. Relationship between lactate production and fructose 2,6-bisphosphate concentration.肝脏糖酵解和糖原合成的葡萄糖敏感性差异。乳酸生成与果糖-2,6-二磷酸浓度之间的关系。
Biochem J. 1984 Dec 15;224(3):779-86. doi: 10.1042/bj2240779.
5
Biochemistry of liver development in the perinatal period.围生期肝脏发育的生物化学
Experientia. 1983 May 15;39(5):473-83. doi: 10.1007/BF01965164.
6
Hepatic carbon flux after re-feeding in the glycogen-storage-disease (gsd/gsd) rat.糖原贮积病(gsd/gsd)大鼠再喂养后的肝脏碳通量。
Biochem J. 1987 Dec 15;248(3):969-72. doi: 10.1042/bj2480969.
7
Continued glucose output after re-feeding contributes to glucose intolerance in hyperthyroidism.再喂养后持续的葡萄糖输出导致甲状腺功能亢进症中的葡萄糖不耐受。
Biochem J. 1987 Nov 1;247(3):801-4. doi: 10.1042/bj2470801.
8
The onset of liver glycogen synthesis in fasted-refed lean and genetically obese (fa/fa) rats.禁食-再喂食的瘦鼠和遗传性肥胖(fa/fa)大鼠肝脏糖原合成的起始
Diabetologia. 1987 Mar;30(3):169-74. doi: 10.1007/BF00274223.
9
Decreased activity and impaired hormonal control of protein phosphatases in rat livers with a deficiency of phosphorylase kinase.磷酸化酶激酶缺乏的大鼠肝脏中蛋白磷酸酶的活性降低及激素调控受损。
Biochem J. 1989 Dec 1;264(2):429-36. doi: 10.1042/bj2640429.