Stefanick M L, Davidson J M
Department of Physiology, Stanford University, CA 94305.
Physiol Behav. 1987;41(5):439-44. doi: 10.1016/0031-9384(87)90078-3.
To determine whether genital dysfunction participates in the sexual inactivity of noncopulator and MPOA-lesioned rats, penile reflexes and spontaneous seminal emission (SSE) were investigated in such animals. The display of penile reflexes was either the same or enhanced in these animals, relative to controls. Noncopulators and MPOA-lesioned rats also did not differ from controls in SSE. Plasma testosterone levels showed no differences between noncopulators and sexually active controls but tended to be elevated in rats bearing MPOA lesions. Finally, SSE did not differ between rats with midthoracic spinal cord transections and controls, suggesting that SSE is not under supraspinal inhibition.
为了确定生殖器功能障碍是否参与了非交配和内侧视前区（MPOA）损伤大鼠的性活动缺乏，对这些动物的阴茎反射和自发性射精（SSE）进行了研究。相对于对照组，这些动物的阴茎反射表现要么相同，要么增强。非交配大鼠和MPOA损伤大鼠在SSE方面也与对照组没有差异。非交配大鼠和性活跃对照组之间的血浆睾酮水平没有差异，但MPOA损伤的大鼠血浆睾酮水平有升高的趋势。最后，胸段脊髓横断大鼠和对照组之间的SSE没有差异，这表明SSE不受脊髓上抑制的影响。
