受MIR156靶向的SPL9被SnRK2s磷酸化并与ABI5相互作用以增强脱落酸反应。（你提供的原文似乎不完整，句末的“in.”后面应该还有具体内容）

MIR156-Targeted SPL9 Is Phosphorylated by SnRK2s and Interacts With ABI5 to Enhance ABA Responses in .

作者信息

Dong Huixue, Yan Suli, Jing Yexing, Yang Ruizhen, Zhang Yunwei, Zhou Yun, Zhu Yingfang, Sun Jiaqiang

机构信息

National Key Facility for Crop Gene Resources and Genetic Improvement, Institute of Crop Sciences, Chinese Academy of Agricultural Sciences, Beijing, China.

State Key Laboratory of Crop Stress Adaptation and Improvement, School of Life Sciences, Henan University, Kaifeng, China.

出版信息

Front Plant Sci. 2021 Jul 21;12:708573. doi: 10.3389/fpls.2021.708573. eCollection 2021.

The miR156-targeted SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) transcription factors play key roles in regulating plant development, but little is known about their function in abscisic acid (ABA) signaling. Here, we report that the miR156-targeted SPLs enhance ABA responses and contribute to the inhibition of pre-harvest sprouting. We find that SPL9 directly activates the expression of ABA responsive genes through binding to their promoters. SPL9 was further shown to physically interact with ABSCISIC ACID INSENSITIVE 5 (ABI5), a master transcription factor in ABA signaling, thus promoting its association with the promoters of ABA responsive genes. Furthermore, we reveal that the protein kinases SnRK2s interact with and phosphorylate SPL9, which is essential for its role in the activation of ABA responses. Together, our results disclose a SnRK2s-SPLs-ABI5 regulatory module in ABA signaling in .

受miR156靶向的SQUAMOSA启动子结合蛋白样（SPL）转录因子在调控植物发育中起关键作用，但它们在脱落酸（ABA）信号传导中的功能却鲜为人知。在此，我们报道受miR156靶向的SPL增强了ABA反应并有助于抑制收获前发芽。我们发现SPL9通过结合ABA反应基因的启动子直接激活其表达。进一步研究表明，SPL9与ABA信号传导中的主要转录因子脱落酸不敏感5（ABI5）发生物理相互作用，从而促进其与ABA反应基因启动子的结合。此外，我们揭示蛋白激酶SnRK2与SPL9相互作用并使其磷酸化，这对其在激活ABA反应中的作用至关重要。总之，我们的结果揭示了植物ABA信号传导中的一个SnRK2-SPL-ABI5调控模块。