Núñez-Samper M, Llanos-Alcázar L F, Viladot-Perice R, Viladot-Voegeli A, Álvarez-Goenaga F, Bailey E J, Parra-Sánchez G, Caldiño-Lozada I, López-Gavito E, Parra-Téllez P
Servicio de Cirugía Ortopédica, Hospital Virgen del Mar. Madrid, España.
Servicio de Cirugía Ortopédica, Hospital 12 de Octubre. Madrid, España.
Acta Ortop Mex. 2021 Jan-Feb;35(1):92-117.
We present the possible etiopatogenic causes of posterior tibial dysfunction or painful flat foot of the adult and the cause-and-effect relationship that may exist. We also expose the gradation of the lesion and the different therapeutic options for the surgical treatment of the deformity. Since 1939, multiple articles have been published, which have been endorsed by clinical, experimental, electromyographic and biomechanical studies; publications that have been consulted and evaluated for the development of this review. In our opinion: the dysfunction of the posterior tibial is caused in principle by a failure of the plantar navicular calcaneus ligament (spring ligament), the main passive stabilizer of the internal plantar arch. This failure would, in time, mean an increase in work of the posterior tibial tendon, in itself "insufficient", which would go into fatigue, until it reached a partial or total rupture. Published work on soft-part procedures acting on the posterior tibial tendon in stage II has not had the expected result in the natural history of deformity. Arthrodesis, on the other hand, has been effective in other stages, but is associated with a loss of movement dynamics in the back foot and increased pressure on adjacent joints.
我们阐述了成人胫后肌腱功能障碍或疼痛性扁平足可能的发病原因以及可能存在的因果关系。我们还揭示了病变的分级以及针对该畸形手术治疗的不同选择。自1939年以来,已发表了多篇文章,这些文章得到了临床、实验、肌电图和生物力学研究的认可;在撰写本综述时参考并评估了这些出版物。我们认为:胫后肌腱功能障碍原则上是由足底舟楔跟韧带(弹簧韧带)失效引起的,该韧带是内侧足底弓的主要被动稳定器。这种失效迟早会导致胫后肌腱本身“力量不足”却需承担更多工作,进而引发疲劳,直至出现部分或完全断裂。已发表的关于在II期对胫后肌腱进行软组织手术的研究,在畸形自然发展过程中并未取得预期效果。另一方面,关节融合术在其他阶段已证明有效,但会导致后足运动动力学丧失以及相邻关节压力增加。