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靶向线粒体的细胞焦亡:创新 COVID-19 疗法的一种有根据的猜测。

Pyroptosis targeting via mitochondria: An educated guess to innovate COVID-19 therapies.

机构信息

Department of Comparative Biomedical Sciences, The Royal Veterinary College, University of London, London, UK.

Department of Biology, University of Rome "Tor Vergata", Rome, Italy.

出版信息

Br J Pharmacol. 2022 May;179(10):2081-2085. doi: 10.1111/bph.15670. Epub 2021 Oct 10.

Abstract

Pyroptosis is a specialized form of inflammatory cell death which aids the defensive response against invading pathogens. Its normally tight regulation is lost during infection by the severe acute respiratory coronavirus 2 (SARS-CoV-2), and thus, uncontrolled pyroptosis disrupts the immune system and the integrity of organs defining the critical conditions in patients with high viral load. Molecular pathways engaged downstream of the formation and stabilization of the inflammasome, which are necessary to execute the process, have been uncovered and drugs are available for their regulation. However, the pharmacology of the upstream events, which are critical to sense and interpret the initial damage by the pathogen, is far from being elucidated. This limits our capacity to identify early markers and targets to ameliorate SARS-CoV-2 linked pyroptosis. Here, we focus attention on the mitochondria and pathways leading to their dysfunction, in order to elucidate the early steps of inflammasome formation and devise tools to predict and counter pathological states induced by SARS-CoV-2. LINKED ARTICLES: This article is part of a themed issue on The second wave: are we any closer to efficacious pharmacotherapy for COVID 19? (BJP 75th Anniversary). To view the other articles in this section visit http://onlinelibrary.wiley.com/doi/10.1111/bph.v179.10/issuetoc.

摘要

细胞焦亡是一种炎症细胞死亡的特殊形式,有助于机体抵抗入侵病原体的防御反应。在严重急性呼吸综合征冠状病毒 2(SARS-CoV-2)感染过程中,其正常的严格调控机制丢失,因此,失控的细胞焦亡会破坏免疫系统和器官的完整性,这是高病毒载量患者出现危急情况的定义特征。下游分子途径的形成和稳定,需要执行该过程,已被揭示,并且存在可用于调节这些途径的药物。但是,对于病原体初始损伤的感应和解释至关重要的上游事件的药理学仍远未阐明。这限制了我们识别早期标志物和靶点以改善 SARS-CoV-2 相关细胞焦亡的能力。在这里,我们关注线粒体及其导致其功能障碍的途径,以阐明细胞焦亡小体形成的早期步骤,并设计工具来预测和对抗 SARS-CoV-2 诱导的病理状态。相关文章:本文是关于《第二波:我们是否更接近有效的 COVID-19 治疗药物?》专题的一部分(BJP 75 周年)。要查看本节中的其他文章,请访问 http://onlinelibrary.wiley.com/doi/10.1111/bph.v179.10/issuetoc.

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