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Heterogeneous NLRP3 inflammasome signature in circulating myeloid cells as a biomarker of COVID-19 severity.循环髓样细胞中异质性 NLRP3 炎性小体特征可作为 COVID-19 严重程度的生物标志物。
Blood Adv. 2021 Mar 9;5(5):1523-1534. doi: 10.1182/bloodadvances.2020003918.
2
Interleukin-6 Is a Biomarker for the Development of Fatal Severe Acute Respiratory Syndrome Coronavirus 2 Pneumonia.白细胞介素-6 是导致致命性严重急性呼吸综合征冠状病毒 2 型肺炎的生物标志物。
Front Immunol. 2021 Feb 18;12:613422. doi: 10.3389/fimmu.2021.613422. eCollection 2021.
3
SARS-CoV-2 engages inflammasome and pyroptosis in human primary monocytes.严重急性呼吸综合征冠状病毒2(SARS-CoV-2)在人原代单核细胞中激活炎性小体并引发细胞焦亡。
Cell Death Discov. 2021 Mar 1;7(1):43. doi: 10.1038/s41420-021-00428-w.
4
Pharmacological advances in mitochondrial therapy.线粒体治疗的药理学进展。
EBioMedicine. 2021 Mar;65:103244. doi: 10.1016/j.ebiom.2021.103244. Epub 2021 Feb 26.
5
Recent updates in the clinical trials of therapeutic monoclonal antibodies targeting cytokine storm for the management of COVID-19.针对细胞因子风暴治疗新冠病毒病(COVID-19)的治疗性单克隆抗体临床试验的最新进展。
Heliyon. 2021 Feb;7(2):e06158. doi: 10.1016/j.heliyon.2021.e06158. Epub 2021 Jan 29.
6
Inflammasome formation in the lungs of patients with fatal COVID-19.COVID-19 致死患者肺部的炎症小体形成。
Inflamm Res. 2021 Jan;70(1):7-10. doi: 10.1007/s00011-020-01413-2. Epub 2020 Oct 20.
7
SARS-CoV-2 triggers inflammatory responses and cell death through caspase-8 activation.SARS-CoV-2 通过半胱天冬酶-8 的激活引发炎症反应和细胞死亡。
Signal Transduct Target Ther. 2020 Oct 9;5(1):235. doi: 10.1038/s41392-020-00334-0.
8
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Inflamm Regen. 2020 Oct 1;40:37. doi: 10.1186/s41232-020-00146-3. eCollection 2020.
9
Inflammasomes and Pyroptosis as Therapeutic Targets for COVID-19.炎症小体和细胞焦亡作为 COVID-19 的治疗靶点。
J Immunol. 2020 Jul 15;205(2):307-312. doi: 10.4049/jimmunol.2000513. Epub 2020 Jun 3.
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FDA-approved disulfiram inhibits pyroptosis by blocking gasdermin D pore formation.美国食品药品监督管理局批准的双硫仑通过阻断gasdermin D 孔形成来抑制细胞焦亡。
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靶向线粒体的细胞焦亡:创新 COVID-19 疗法的一种有根据的猜测。

Pyroptosis targeting via mitochondria: An educated guess to innovate COVID-19 therapies.

机构信息

Department of Comparative Biomedical Sciences, The Royal Veterinary College, University of London, London, UK.

Department of Biology, University of Rome "Tor Vergata", Rome, Italy.

出版信息

Br J Pharmacol. 2022 May;179(10):2081-2085. doi: 10.1111/bph.15670. Epub 2021 Oct 10.

DOI:10.1111/bph.15670
PMID:34632567
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8653109/
Abstract

Pyroptosis is a specialized form of inflammatory cell death which aids the defensive response against invading pathogens. Its normally tight regulation is lost during infection by the severe acute respiratory coronavirus 2 (SARS-CoV-2), and thus, uncontrolled pyroptosis disrupts the immune system and the integrity of organs defining the critical conditions in patients with high viral load. Molecular pathways engaged downstream of the formation and stabilization of the inflammasome, which are necessary to execute the process, have been uncovered and drugs are available for their regulation. However, the pharmacology of the upstream events, which are critical to sense and interpret the initial damage by the pathogen, is far from being elucidated. This limits our capacity to identify early markers and targets to ameliorate SARS-CoV-2 linked pyroptosis. Here, we focus attention on the mitochondria and pathways leading to their dysfunction, in order to elucidate the early steps of inflammasome formation and devise tools to predict and counter pathological states induced by SARS-CoV-2. LINKED ARTICLES: This article is part of a themed issue on The second wave: are we any closer to efficacious pharmacotherapy for COVID 19? (BJP 75th Anniversary). To view the other articles in this section visit http://onlinelibrary.wiley.com/doi/10.1111/bph.v179.10/issuetoc.

摘要

细胞焦亡是一种炎症细胞死亡的特殊形式,有助于机体抵抗入侵病原体的防御反应。在严重急性呼吸综合征冠状病毒 2(SARS-CoV-2)感染过程中,其正常的严格调控机制丢失,因此,失控的细胞焦亡会破坏免疫系统和器官的完整性,这是高病毒载量患者出现危急情况的定义特征。下游分子途径的形成和稳定,需要执行该过程,已被揭示,并且存在可用于调节这些途径的药物。但是,对于病原体初始损伤的感应和解释至关重要的上游事件的药理学仍远未阐明。这限制了我们识别早期标志物和靶点以改善 SARS-CoV-2 相关细胞焦亡的能力。在这里,我们关注线粒体及其导致其功能障碍的途径,以阐明细胞焦亡小体形成的早期步骤,并设计工具来预测和对抗 SARS-CoV-2 诱导的病理状态。相关文章:本文是关于《第二波:我们是否更接近有效的 COVID-19 治疗药物?》专题的一部分(BJP 75 周年)。要查看本节中的其他文章,请访问 http://onlinelibrary.wiley.com/doi/10.1111/bph.v179.10/issuetoc.