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1
Structural alterations of DNA ligase I in Bloom syndrome.
Proc Natl Acad Sci U S A. 1987 Nov;84(22):8016-20. doi: 10.1073/pnas.84.22.8016.
2
Biochemical properties of mammalian DNA ligase I and the molecular defect in Bloom's syndrome.
Prog Clin Biol Res. 1990;340A:283-94.
3
DNA ligase I, Bloom's syndrome, and cancer.
Cancer Genet Cytogenet. 1988 Jan;30(1):181-2. doi: 10.1016/0165-4608(88)90110-0.
4
Expression of a DNA-ligase-stimulatory factor in Bloom's syndrome cell line GM1492.
Eur J Biochem. 1988 Jun 15;174(3):465-70. doi: 10.1111/j.1432-1033.1988.tb14121.x.
5
DNA ligase I deficiency in Bloom's syndrome.
Nature. 1987;325(6102):355-7. doi: 10.1038/325355a0.
6
Concomitant reversion of the characteristic phenotypic properties of a cell line of Bloom's syndrome origin.
Carcinogenesis. 1989 Jan;10(1):217-9. doi: 10.1093/carcin/10.1.217.
7
Defective DNA ligase I in Bloom's syndrome cells. Simultaneous analysis using immunoblotting and the ligase-[32P]AMP adduct assay.
J Biol Chem. 1988 Dec 5;263(34):18231-5.
8
Altered DNA ligase I activity in Bloom's syndrome cells.
Nature. 1987;325(6102):357-9. doi: 10.1038/325357a0.
9
Phenotypic correction of a human cell line (46BR) with aberrant DNA ligase I activity.
Mutat Res. 1993 Jun;294(1):51-8. doi: 10.1016/0921-8777(93)90057-n.
10
Two types of DNA ligase I activity in lymphoblastoid cells from patients with Bloom's syndrome.
Jpn J Cancer Res. 1991 Jan;82(1):51-7. doi: 10.1111/j.1349-7006.1991.tb01745.x.
引用本文的文献
1
is a novel marker for bladder cancer prognosis: evidence based on experimental studies, machine learning and single-cell sequencing.
Front Immunol. 2024 Aug 21;15:1419126. doi: 10.3389/fimmu.2024.1419126. eCollection 2024.
2
No relationship between genetic instability in Bloom's syndrome and DNA hypomethylation of some major repetitive sequences.
Hum Genet. 1993 Aug;92(1):57-60. doi: 10.1007/BF00216145.
3
DNA ligase III is the major high molecular weight DNA joining activity in SV40-transformed human fibroblasts: normal levels of DNA ligase III activity in Bloom syndrome cells.
Nucleic Acids Res. 1993 Nov 25;21(23):5425-30. doi: 10.1093/nar/21.23.5425.
4
An interaction between the mammalian DNA repair protein XRCC1 and DNA ligase III.
Mol Cell Biol. 1994 Jan;14(1):68-76. doi: 10.1128/mcb.14.1.68-76.1994.
5
Increased rate of spontaneous mitotic recombination in T lymphocytes from a Bloom's syndrome patient using a flow-cytometric assay at HLA-A locus.
Jpn J Cancer Res. 1994 Jun;85(6):610-8. doi: 10.1111/j.1349-7006.1994.tb02403.x.
6
DNA ligase I mediates essential functions in mammalian cells.
Mol Cell Biol. 1995 Aug;15(8):4303-8. doi: 10.1128/MCB.15.8.4303.
7
Mammalian DNA ligase III: molecular cloning, chromosomal localization, and expression in spermatocytes undergoing meiotic recombination.
Mol Cell Biol. 1995 Oct;15(10):5412-22. doi: 10.1128/MCB.15.10.5412.
8
9
Bloom syndrome: a single complementation group defines patients of diverse ethnic origin.
Am J Hum Genet. 1988 Jun;42(6):816-24.
10
High frequency of large spontaneous deletions of DNA in tumor-derived CHEF cells.
Proc Natl Acad Sci U S A. 1989 Apr;86(7):2306-10. doi: 10.1073/pnas.86.7.2306.
本文引用的文献
1
A method for determining the sedimentation behavior of enzymes: application to protein mixtures.
J Biol Chem. 1961 May;236:1372-9.
2
Congenital telangiectatic erythema resembling lupus erythematosus in dwarfs; probably a syndrome entity.
AMA Am J Dis Child. 1954 Dec;88(6):754-8.
3
Removal of O6-methylguanine from DNA of normal and xeroderma pigmentosum-derived lymphoblastoid lines.
Nature. 1981 Jan 29;289(5796):417-20. doi: 10.1038/289417a0.
4
DNA repair in lymphoblastoid cell lines established from human genetic disorders.
Chem Biol Interact. 1980 Dec;33(1):63-81. doi: 10.1016/0009-2797(80)90044-7.
5
Bloom's syndrome in a Japanese girl.
Clin Genet. 1980 Feb;17(2):143-8. doi: 10.1111/j.1399-0004.1980.tb00123.x.
6
Human tumor strains defective in the repair of alkylated DNA fail to regenerate rapidly-sedimenting nucleoids after N-methyl-N'-nitro-N-nitrosoguanidine treatment.
Carcinogenesis. 1981;2(11):1215-8. doi: 10.1093/carcin/2.11.1215.
7
Tendency to high levels of UVR-induced unscheduled DNA synthesis in Bloom syndrome.
Mutat Res. 1981 Apr;81(2):229-41. doi: 10.1016/0027-5107(81)90037-3.
8
Formation of DNA single-strand breaks by near-ultraviolet and gamma-rays in normal and Bloom's syndrome skin fibroblasts.
Cancer Res. 1981 May;41(5):2003-7.
9
DNA fork displacement rates in Bloom's syndrome fibroblasts.
Biochim Biophys Acta. 1982 Feb 26;696(2):226-7. doi: 10.1016/0167-4781(82)90034-3.
10
Elevated spontaneous mutation rate in Bloom syndrome fibroblasts.
Proc Natl Acad Sci U S A. 1981 May;78(5):3133-7. doi: 10.1073/pnas.78.5.3133.
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