Fasanaro A M, Spitaleri D L, Valiani R, Postiglione A, Soricelli A, Mansi L, Grossi D
Divisione di Neurologia, Ospedale A. Cardarelli, Napoli, Italy.
J Neurol. 1987 Aug;234(6):421-3. doi: 10.1007/BF00314089.
A 59-year-old man is reported, who became aphasic after left thalamic infarction, shown by CT. His speech was fluent, with reduced voice volume, impaired auditory and reading comprehension, verbal paraphasias but intact repetition skills. A single photon emission computed tomography (SPECT) scan to measure regional cerebral flow (rCBF) showed a reduction of flow in the parietotemporal areas of the left hemisphere. It is suggested that thalamic aphasia could result from structural subcortical damage with a homolateral functional cortical deficit leading to the specific aphasic disturbance.
据报道，一名59岁男性在左侧丘脑梗死（CT显示）后出现失语。他的言语流畅，但音量降低，听觉和阅读理解受损，存在言语错乱，但复述能力完好。通过单光子发射计算机断层扫描（SPECT）测量局部脑血流量（rCBF）显示，左半球顶颞区域血流量减少。提示丘脑性失语可能是由于皮质下结构损伤伴同侧功能性皮质缺损导致特定的失语障碍。
