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TOM70 in Glial Cells as a Potential Target for Treatment of COVID-19.

作者信息

Montenegro Yorran Hardman Araújo, Zanatta Geancarlo, Quincozes-Santos André, Leipnitz Guilhian

机构信息

Programa de Pós-Graduação em Neurociências, Universidade Federal do Rio Grande do Sul, Porto Alegre, Brazil.

Programa de Pós-Graduação em Bioquímica, Universidade Federal do Ceará, Fortaleza, Brazil.

出版信息

Front Cell Neurosci. 2021 Dec 24;15:811376. doi: 10.3389/fncel.2021.811376. eCollection 2021.

Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/a2ed/8740195/b3345c8b72ef/fncel-15-811376-g0001.jpg

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本文引用的文献

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SARS-CoV-2 infection impacts carbon metabolism and depends on glutamine for replication in Syrian hamster astrocytes.
J Neurochem. 2022 Oct;163(2):113-132. doi: 10.1111/jnc.15679. Epub 2022 Aug 15.
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SARS-CoV-2-specific neuropathology: fact or fiction?
Trends Neurosci. 2021 Dec;44(12):933-935. doi: 10.1016/j.tins.2021.10.006. Epub 2021 Oct 16.
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The SARS-CoV-2 main protease M causes microvascular brain pathology by cleaving NEMO in brain endothelial cells.
Nat Neurosci. 2021 Nov;24(11):1522-1533. doi: 10.1038/s41593-021-00926-1. Epub 2021 Oct 21.
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Acute and chronic neurological disorders in COVID-19: potential mechanisms of disease.
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Neurochemical signs of astrocytic and neuronal injury in acute COVID-19 normalizes during long-term follow-up.
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Expression of SARS-CoV-2-related receptors in cells of the neurovascular unit: implications for HIV-1 infection.
J Neuroinflammation. 2021 Jul 29;18(1):167. doi: 10.1186/s12974-021-02210-2.
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Evolution of enhanced innate immune evasion by the SARS-CoV-2 B.1.1.7 UK variant.
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