进行性系统性硬化症（硬皮病）患者的真皮成纤维细胞培养物中胶原蛋白积累增加。 - Suppr

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超能文献

Increased collagen accumulation in dermal fibroblast cultures from patients with progressive systemic sclerosis (scleroderma).

作者信息

Buckingham R B, Prince R K, Rodnan G P, Taylor F

出版信息

J Lab Clin Med. 1978 Jul;92(1):5-21.

PMID:351108

Abstract

摘要

相似文献

Increased collagen accumulation in dermal fibroblast cultures from patients with progressive systemic sclerosis (scleroderma).进行性系统性硬化症（硬皮病）患者的真皮成纤维细胞培养物中胶原蛋白积累增加。

J Lab Clin Med. 1978 Jul;92(1):5-21.

Progressive systemic sclerosis (PSS, scleroderma) dermal fibroblasts synthesize increased amounts of glycosaminoglycan.进行性系统性硬化症（PSS，硬皮病）的真皮成纤维细胞合成的糖胺聚糖量增加。

J Lab Clin Med. 1983 May;101(5):659-69.

Expression of osteonectin, decorin, and transforming growth factor-beta 1 genes in fibroblasts cultured from patients with systemic sclerosis and morphea.骨连接蛋白、核心蛋白聚糖和转化生长因子-β1基因在系统性硬化症和硬斑病患者培养的成纤维细胞中的表达

J Rheumatol. 1991 Feb;18(2):247-51.

[Characteristics of collagen protein biosynthesis in fibroblast cultures from the skin of patients with systemic scleroderma].[系统性硬皮病患者皮肤成纤维细胞培养中胶原蛋白生物合成的特征]

Vopr Revm. 1981 Apr-Jun(2):44-50.

Products of activated mononuclear cells modulate accumulation of collagen by normal dermal and scleroderma fibroblasts in culture.活化单核细胞的产物可调节培养的正常真皮成纤维细胞和硬皮病成纤维细胞中胶原蛋白的积累。

J Lab Clin Med. 1984 Sep;104(3):355-69.

Expression of interleukin-4 in scleroderma skin specimens and scleroderma fibroblast cultures. Potential role in fibrosis.白细胞介素-4在硬皮病皮肤标本及硬皮病成纤维细胞培养物中的表达。在纤维化中的潜在作用。

Arch Dermatol. 1996 Jul;132(7):802-6.

Gamma-interferon inhibits extracellular matrix synthesis and remodeling in collagen lattice cultures of normal and scleroderma skin fibroblasts.γ-干扰素抑制正常和硬皮病皮肤成纤维细胞胶原晶格培养物中的细胞外基质合成和重塑。

Eur J Cell Biol. 1992 Apr;57(2):244-53.

The characterization of normal and scleroderma skin fibroblasts cultured in a collagen gel matrix.在胶原凝胶基质中培养的正常皮肤成纤维细胞和硬皮病皮肤成纤维细胞的特性研究。

J Rheumatol. 1988 Feb;15(2):268-75.

[Effect of isoproterenol on collagen synthesis in cultured skin fibroblasts in patients with systemic scleroderma].

Vopr Med Khim. 1984;30(6):92-4.

[Collagen metabolism in systemic scleroderma].

Vopr Med Khim. 1978 Nov-Dec;24(6):789-95.

引用本文的文献

Cellular Transdifferentiation: A Crucial Mechanism of Fibrosis in Systemic Sclerosis.细胞转分化：系统性硬化症纤维化的关键机制。

Curr Rheumatol Rev. 2024;20(4):388-404. doi: 10.2174/0115733971261932231025045400.

Proteomic analysis identification of a pattern of shared alterations in the secretome of dermal fibroblasts from systemic sclerosis and nephrogenic systemic fibrosis.蛋白质组学分析鉴定系统性硬化症和肾源性系统性纤维化皮肤成纤维细胞外泌体中共同改变的模式。

Am J Pathol. 2010 Oct;177(4):1638-46. doi: 10.2353/ajpath.2010.091095. Epub 2010 Aug 19.

Histomorphometric analysis of cutaneous remodeling in the early stage of the scleroderma model.硬皮病模型早期皮肤重塑的组织形态计量学分析。

Clinics (Sao Paulo). 2009;64(6):577-83. doi: 10.1590/s1807-59322009000600014.

Interstitial lung disease in systemic sclerosis.系统性硬化症中的间质性肺疾病

Lung. 2007 Jul-Aug;185(4):211-20. doi: 10.1007/s00408-007-9012-3.

Increased expression of integrin alphavbeta5 induces the myofibroblastic differentiation of dermal fibroblasts.整合素αvβ5表达增加诱导真皮成纤维细胞向肌成纤维细胞分化。

Am J Pathol. 2006 Feb;168(2):499-510. doi: 10.2353/ajpath.2006.041306.

Increased expression levels of integrin alphavbeta5 on scleroderma fibroblasts.硬皮病成纤维细胞上整合素αvβ5表达水平升高。

Am J Pathol. 2004 Apr;164(4):1275-92. doi: 10.1016/s0002-9440(10)63215-4.

Impaired Smad7-Smurf-mediated negative regulation of TGF-beta signaling in scleroderma fibroblasts.硬皮病成纤维细胞中Smad7-Smurf介导的TGF-β信号负调控受损。

J Clin Invest. 2004 Jan;113(2):253-64. doi: 10.1172/JCI16269.

Endogenous IL-1alpha from systemic sclerosis fibroblasts induces IL-6 and PDGF-A.系统性硬化症成纤维细胞产生的内源性白细胞介素-1α可诱导白细胞介素-6和血小板衍生生长因子-A。

J Clin Invest. 1999 May;103(9):1253-60. doi: 10.1172/JCI4304.

Differential in vitro responses of elastin expression to basic fibroblast growth factor and transforming growth factor beta 1 in upper, middle and lower dermal fibroblasts.

Arch Dermatol Res. 1996 Nov;288(12):753-6. doi: 10.1007/BF02505292.

Progressive systemic sclerosis of the internal anal sphincter leading to passive faecal incontinence.导致被动性大便失禁的内括约肌进行性系统性硬化症。

Gut. 1994 Jun;35(6):857-9. doi: 10.1136/gut.35.6.857.

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