Takei Seira, Taketomi Shuji, Torii Suguru, Tojima Michio, Kaneoka Koji, Tanaka Sakae
Department of Orthopaedic Surgery, Sensory and Motor System Medicine, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.
Waseda Institute of Human Growth and Development, Waseda University, Saitama, Japan.
Orthop J Sports Med. 2022 Mar 14;10(3):23259671221083567. doi: 10.1177/23259671221083567. eCollection 2022 Mar.
Osgood-Schlatter disease (OSD) is an apophysitis of the tibial tubercle caused by repeated traction of the patellar tendon during adolescence. Although OSD is associated more with sports such as soccer, it remains unclear whether the kicking motion itself is related to OSD onset.
To prospectively evaluate the kicking motion in adolescent soccer players who later developed OSD.
Case-control study.
The authors observed 47 Japanese adolescent male soccer players (mean age, 12 years) over 6 months in 2018-2019; none of the participants had OSD at study onset. The players underwent tibial tubercle ultrasound at baseline, and their kicking motion was evaluated using 3-dimensional (3D) motion analysis with 65 reflective markers. The 3D angle of the lumbar spine, thorax, and pelvis and the angular velocity of the hips, knees, and ankles were calculated for the support leg. The kicking motion was divided into 8 phases, and ball speed, kicking time, and positions of the support (nonkicking) leg and center of mass (COM) from the ball were calculated for each phase. Six months later, the players underwent another ultrasound and were divided into 2 groups: those diagnosed with OSD in the support leg (OSD group) and those without OSD (normal [NRL] group). All factors calculated using the kicking motion analysis at the baseline were compared between groups.
There were 19 players in the OSD group and 28 players in the NRL group. Anthropometric measurements, ball speed, and kicking time were similar between the 2 groups. The forward translation of the COM and the support leg, the flexion angle of the thorax, and the rotation angle of the pelvis before impact with the ball were all significantly smaller in the OSD group than in the NRL group ( < .05). Moreover, the knee extension angular velocity of the support leg was significantly larger in the OSD group ( < .05) at ball impact.
A kicking motion with small COM translation, small thoracic flexion, and small pelvic rotation before ball impact, as well as large knee extension angular velocity of the support leg at ball impact, may be associated with OSD onset.
奥斯古德-施拉特病(OSD)是青少年时期髌腱反复牵拉导致的胫骨结节骨突炎。尽管OSD更多地与足球等运动相关,但尚不清楚踢球动作本身是否与OSD的发病有关。
前瞻性评估后来发生OSD的青少年足球运动员的踢球动作。
病例对照研究。
作者在2018 - 2019年的6个月内观察了47名日本青少年男性足球运动员(平均年龄12岁);研究开始时所有参与者均无OSD。球员在基线时接受胫骨结节超声检查,并使用带有65个反光标记的三维(3D)运动分析评估他们的踢球动作。计算支撑腿的腰椎、胸部和骨盆的3D角度以及髋、膝和踝关节的角速度。踢球动作分为8个阶段,并计算每个阶段的球速、踢球时间以及支撑(非踢球)腿和质心(COM)相对于球的位置。6个月后,球员再次接受超声检查,并分为两组:支撑腿被诊断为OSD的球员(OSD组)和无OSD的球员(正常[NRL]组)。比较两组在基线时使用踢球动作分析计算的所有因素。
OSD组有19名球员,NRL组有28名球员。两组的人体测量数据、球速和踢球时间相似。在球撞击前,OSD组的COM和支撑腿的向前平移、胸部的屈曲角度以及骨盆的旋转角度均显著小于NRL组(P <.05)。此外,在球撞击时,OSD组支撑腿的膝关节伸展角速度显著更大(P <.0