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An Airway-Centric View of Idiopathic Pulmonary Fibrosis.

作者信息

Stancil Ian T, Michalski Jacob E, Schwartz David A

机构信息

Department of Medicine and.

Department of Immunology and Microbiology, University of Colorado Anschutz Medical Campus, Aurora, Colorado.

出版信息

Am J Respir Crit Care Med. 2022 Aug 15;206(4):410-416. doi: 10.1164/rccm.202109-2219PP.

DOI:10.1164/rccm.202109-2219PP
PMID:35446237
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12039158/
Abstract
摘要

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Integrin Axis Regulates Airway Biophysical Dysfunction in Idiopathic Pulmonary Fibrosis.整合素轴调节特发性肺纤维化中的气道生物物理功能障碍。
Am J Respir Cell Mol Biol. 2022 Feb;66(2):235-237. doi: 10.1165/rcmb.2021-0224LE.
2
Human alveolar type 2 epithelium transdifferentiates into metaplastic KRT5 basal cells.人肺泡 II 型上皮细胞转分化为 KRT5 基底细胞样的化生细胞。
Nat Cell Biol. 2022 Jan;24(1):10-23. doi: 10.1038/s41556-021-00809-4. Epub 2021 Dec 30.
3
Molecular programs of fibrotic change in aging human lung.衰老人肺纤维化改变的分子程序。
Nat Commun. 2021 Nov 2;12(1):6309. doi: 10.1038/s41467-021-26603-2.
4
Small Airway Reduction and Fibrosis Is an Early Pathologic Feature of Idiopathic Pulmonary Fibrosis.小气道减少和纤维化是特发性肺纤维化的早期病理特征。
Am J Respir Crit Care Med. 2021 Nov 1;204(9):1048-1059. doi: 10.1164/rccm.202103-0585OC.
5
Pulmonary fibrosis distal airway epithelia are dynamically and structurally dysfunctional.肺纤维化导致远端气道上皮细胞的动态和结构功能障碍。
Nat Commun. 2021 Jul 27;12(1):4566. doi: 10.1038/s41467-021-24853-8.
6
Molecular mapping of interstitial lung disease reveals a phenotypically distinct senescent basal epithelial cell population.分子图谱分析揭示了一种表型独特的衰老基底上皮细胞群体。
JCI Insight. 2021 Apr 22;6(8):143626. doi: 10.1172/jci.insight.143626.
7
Genetic Risk Factors for Idiopathic Pulmonary Fibrosis: Insights into Immunopathogenesis.特发性肺纤维化的遗传风险因素：对免疫发病机制的见解
J Inflamm Res. 2021 Jan 5;13:1305-1318. doi: 10.2147/JIR.S280958. eCollection 2020.
8
Molecular markers of telomere dysfunction and senescence are common findings in the usual interstitial pneumonia pattern of lung fibrosis.端粒功能障碍和衰老的分子标志物在肺纤维化的寻常型间质性肺炎模式中很常见。
Histopathology. 2021 Jul;79(1):67-76. doi: 10.1111/his.14334. Epub 2021 Apr 14.
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Regionalized tissue fluidization is required for epithelial gap closure during insect gastrulation.区域组织化的组织液流对于昆虫原肠胚形成过程中上皮细胞间隙的闭合是必需的。
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Gli1 mesenchymal stromal cells form a pathological niche to promote airway progenitor metaplasia in the fibrotic lung.Gli1 间质基质细胞形成病理性小生境，促进纤维肺中的气道祖细胞化生。
Nat Cell Biol. 2020 Nov;22(11):1295-1306. doi: 10.1038/s41556-020-00591-9. Epub 2020 Oct 12.