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腺相关病毒 9 介导的柯萨奇病毒 B3 诱导心肌炎中心肌保护细胞因子的功能筛选。

AAV9-mediated functional screening for cardioprotective cytokines in Coxsackievirus-B3-induced myocarditis.

机构信息

Department of Cardiovascular Sciences, Center for Vascular and Molecular Biology, KU Leuven, Leuven, Belgium.

CARIM, Maastricht University, Maastricht, The Netherlands.

出版信息

Sci Rep. 2022 May 4;12(1):7304. doi: 10.1038/s41598-022-11131-w.

DOI:10.1038/s41598-022-11131-w
PMID:35508525
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9067557/
Abstract

Viral myocarditis (VM) is an important cause of heart failure (HF) in children and adults. However, the molecular determinants involved in cardiac inflammation and cardiomyocyte necrosis remain poorly characterized, and cardioprotective molecules are currently missing. Here, we applied an in vivo method based on the functional selection (FunSel) of cardioprotective factors using AAV vectors for the unbiased identification of novel immunomodulatory molecules in a Coxsackievirus B3 (CVB3)-induced myocarditis mouse model. Two consecutive rounds of in vivo FunSel using an expression library of 60 cytokines were sufficient to identify five cardioprotective factors (IL9, IL3, IL4, IL13, IL15). The screening also revealed three cytokines (IL18, IL17b, and CCL11) that were counter-selected and likely to exert a detrimental effect. The pooled overexpression of the five most enriched cytokines using AAV9 vectors decreased inflammation and reduced cardiac dilatation, persisting at 1 month after treatment. Individual overexpression of IL9, the top ranking in our functional selection, markedly reduced cardiac inflammation and injury, concomitant with an increase of anti-inflammatory Th2-cells and a reduction of pro-inflammatory Th17- and Th22-cells at 14 days post-infection. AAV9-mediated FunSel cardiac screening identified IL9 and other four cytokines (IL3, IL4, IL13, and IL15) as cardioprotective factors in CVB3-induced VM in mice.

摘要

病毒性心肌炎 (VM) 是儿童和成人心力衰竭 (HF) 的重要病因。然而,涉及心脏炎症和心肌细胞坏死的分子决定因素仍未得到很好的描述,并且目前缺乏心脏保护分子。在这里,我们应用了一种基于使用 AAV 载体对心脏保护因子进行功能选择 (FunSel) 的体内方法,用于在柯萨奇病毒 B3 (CVB3) 诱导的心肌炎小鼠模型中鉴定新型免疫调节分子。使用 60 种细胞因子表达文库进行两轮连续的体内 FunSel 足以鉴定出五种心脏保护因子 (IL9、IL3、IL4、IL13 和 IL15)。筛选还揭示了三种可能发挥有害作用的被反选的细胞因子 (IL18、IL17b 和 CCL11)。使用 AAV9 载体过表达五种最丰富的细胞因子可减少炎症并减轻心脏扩张,在治疗后 1 个月仍持续存在。在感染后 14 天,我们功能选择中排名第一的 IL9 的过表达可显著减少心脏炎症和损伤,同时增加抗炎性 Th2 细胞并减少促炎性 Th17 和 Th22 细胞。AAV9 介导的 FunSel 心脏筛选鉴定出 IL9 和其他四种细胞因子 (IL3、IL4、IL13 和 IL15) 作为 CVB3 诱导的 VM 中的心脏保护因子。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/a951005ae6be/41598_2022_11131_Fig8_HTML.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/0412a9a33fb1/41598_2022_11131_Fig4_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/b5315b51d38d/41598_2022_11131_Fig5_HTML.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/abe075ddfefb/41598_2022_11131_Fig7_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/a951005ae6be/41598_2022_11131_Fig8_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/38b309018fea/41598_2022_11131_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/9b60a3e9118d/41598_2022_11131_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/38791c576297/41598_2022_11131_Fig3_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/0412a9a33fb1/41598_2022_11131_Fig4_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/b5315b51d38d/41598_2022_11131_Fig5_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/4ea103a912c7/41598_2022_11131_Fig6_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/abe075ddfefb/41598_2022_11131_Fig7_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2472/9068805/a951005ae6be/41598_2022_11131_Fig8_HTML.jpg

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