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Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD.毛细血管停滞:AD/ADRD中脑血流量减少的一种机制
J Exp Neurol. 2021;2(4):149-153. doi: 10.33696/neurol.2.048.
2
Influence of dual-specificity protein phosphatase 5 on mechanical properties of rat cerebral and renal arterioles.双重特异性蛋白磷酸酶 5 对大鼠脑和肾小动脉力学特性的影响。
Physiol Rep. 2020 Jan;8(2):e14345. doi: 10.14814/phy2.14345.

从 1901 年到 2022 年,我们离真正了解与年龄相关的痴呆症的发病机制还有多远?

From 1901 to 2022, how far are we from truly understanding the pathogenesis of age-related dementia?

机构信息

Department of Pharmacology and Toxicology, University of Mississippi Medical Center, 2500 North State Street, Jackson, MS, 39216, USA.

出版信息

Geroscience. 2022 Jun;44(3):1879-1883. doi: 10.1007/s11357-022-00591-7. Epub 2022 May 19.

DOI:10.1007/s11357-022-00591-7
PMID:35585301
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9213583/
Abstract

From the first described AD case in 1901 to the current year 2022, understanding the pathogenesis of Alzheimer's disease (AD) and dementia has undergone a long and tortuous journey. Many mechanisms of AD etiology have been proposed and studied. However, current medications and FDA-approved treatments cannot cure AD and AD-related dementias (AD/ADRD). Recently, brain hypoperfusion associated with neurovascular dysfunction was recognized as one of the causal factors in the development of AD dementia. Arteriosclerotic changes were observed in the first AD case. A recent study reported that the functional hyperemic response to whisker stimulation was reduced in 9-12 months old atherosclerotic mice. Interestingly, they found that evoked hemodynamic responses were not altered in age-matched AD mice or AD mice with superimposed atherosclerosis using 2D-optical imaging spectroscopy in chronic studies. However, functional hyperemia was impaired in AD mice using the same approach in an acute study. It is essential to scrutinize the available data critically since different genetic backgrounds, ages, sexes of studied animal models, and different approaches used for the same function even structural examination may provide opposite information. We certainly are closer to truly understanding the pathogenesis of dementia. We expect positive results from using aducanumab (Aduhelm®) as the first FDA-approved anti-amyloid monoclonal antibody as a treatment for AD/ADRD. We hope to identify and develop new drugs targeting other potential contributing mechanisms such as the cerebral vascular pathways.

摘要

从 1901 年首例 AD 病例描述至今 2022 年,对阿尔茨海默病 (AD) 和痴呆症发病机制的理解经历了漫长而曲折的历程。许多 AD 病因机制已被提出并进行了研究。然而,目前的药物和 FDA 批准的治疗方法无法治愈 AD 和 AD 相关痴呆症 (AD/ADRD)。最近,与神经血管功能障碍相关的脑灌注不足被认为是 AD 痴呆发展的原因之一。在首例 AD 病例中观察到动脉粥样硬化变化。最近的一项研究报告称,在 9-12 个月大的动脉粥样硬化小鼠中,胡须刺激的功能性充血反应减少。有趣的是,他们发现,在慢性研究中使用 2D 光学成像光谱学,在年龄匹配的 AD 小鼠或叠加动脉粥样硬化的 AD 小鼠中,诱发的血流动力学反应没有改变。然而,在急性研究中,使用相同方法发现 AD 小鼠的功能性充血受损。仔细审查现有数据至关重要,因为不同的遗传背景、研究动物模型的年龄、性别以及用于同一功能甚至结构检查的不同方法可能会提供相反的信息。我们肯定更接近于真正理解痴呆症的发病机制。我们期望使用 aducanumab (Aduhelm®) 作为首个 FDA 批准的抗淀粉样蛋白单克隆抗体治疗 AD/ADRD 会带来积极的结果。我们希望确定并开发针对其他潜在致病机制(如脑血管途径)的新药。