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Air pollution, bronchiolitis, and asthma: the role of nasal microRNAs.

作者信息

Makrinioti Heidi, Camargo Carlos A, Zhu Zhaozhong, Freishtat Robert J, Hasegawa Kohei

机构信息

London School of Paediatrics, London, UK.

Department of Emergency Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.

出版信息

Lancet Respir Med. 2022 Aug;10(8):733-734. doi: 10.1016/S2213-2600(22)00133-3. Epub 2022 May 17.

DOI:10.1016/S2213-2600(22)00133-3
PMID:35594872
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9339524/
Abstract
摘要

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Severe bronchiolitis profiles and risk of asthma development in Finnish children.严重细支气管炎特征与芬兰儿童哮喘发病风险。
J Allergy Clin Immunol. 2022 Apr;149(4):1281-1285.e1. doi: 10.1016/j.jaci.2021.08.035. Epub 2021 Oct 5.
2
Proximity to Major Roads and Risks of Childhood Recurrent Wheeze and Asthma in a Severe Bronchiolitis Cohort.主干道附近与严重毛细支气管炎队列儿童反复喘息和哮喘风险。
Int J Environ Res Public Health. 2021 Apr 15;18(8):4197. doi: 10.3390/ijerph18084197.
3
The epigenetic pioneer EGR2 initiates DNA demethylation in differentiating monocytes at both stable and transient binding sites.表观遗传先驱 EGR2 在分化中的单核细胞中启动稳定和瞬时结合位点的 DNA 去甲基化。
Nat Commun. 2021 Mar 10;12(1):1556. doi: 10.1038/s41467-021-21661-y.
4
Exosomal miRNA-19a and miRNA-614 Induced by Air Pollutants Promote Proinflammatory M1 Macrophage Polarization via Regulation of RORα Expression in Human Respiratory Mucosal Microenvironment.空气污染物诱导的外泌体miRNA-19a和miRNA-614通过调节人呼吸道黏膜微环境中RORα的表达促进促炎M1巨噬细胞极化。
J Immunol. 2020 Dec 1;205(11):3179-3190. doi: 10.4049/jimmunol.2000456. Epub 2020 Oct 28.
5
The nasal methylome as a biomarker of asthma and airway inflammation in children.鼻腔甲基组作为儿童哮喘和气道炎症的生物标志物。
Nat Commun. 2019 Jul 12;10(1):3095. doi: 10.1038/s41467-019-11058-3.
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Gene activation precedes DNA demethylation in response to infection in human dendritic cells.基因激活先于 DNA 去甲基化，以响应人树突状细胞中的感染。
Proc Natl Acad Sci U S A. 2019 Apr 2;116(14):6938-6943. doi: 10.1073/pnas.1814700116. Epub 2019 Mar 18.
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Pediatr Res. 2018 Mar;83(3):606-614. doi: 10.1038/pr.2017.309. Epub 2018 Jan 17.
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Antagonism of microRNA-126 suppresses the effector function of TH2 cells and the development of allergic airways disease.微小RNA-126的拮抗作用可抑制TH2细胞的效应功能及变应性气道疾病的发展。
Proc Natl Acad Sci U S A. 2009 Nov 3;106(44):18704-9. doi: 10.1073/pnas.0905063106. Epub 2009 Oct 20.