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糖尿病血管病变的演变。

Evolution and diabetic vasculopathy.

机构信息

Komatsu University, Komatsu, Japan.

Department of Biochemistry and Molecular Vascular Biology, Kanazawa University Graduate School of Medical Sciences, Kanazawa, Japan.

出版信息

J Diabetes Investig. 2022 Jul;13(7):1111-1113. doi: 10.1111/jdi.13843. Epub 2022 Jun 9.

DOI:10.1111/jdi.13843
PMID:35598317
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9248414/
Abstract

Evolution of blood sugar, glycation, receptor for advanced glycation end-products and diabetic vasculopathy.

摘要

血糖、糖基化、晚期糖基化终产物受体和糖尿病血管病变的演变。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/22da/9248414/2fbdd6616e8e/JDI-13-1111-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/22da/9248414/2fbdd6616e8e/JDI-13-1111-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/22da/9248414/2fbdd6616e8e/JDI-13-1111-g002.jpg

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1
Evolution and diabetic vasculopathy.糖尿病血管病变的演变。
J Diabetes Investig. 2022 Jul;13(7):1111-1113. doi: 10.1111/jdi.13843. Epub 2022 Jun 9.
2
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The receptor for advanced glycation end-products has a central role in mediating the effects of advanced glycation end-products on the development of vascular disease in diabetes mellitus.晚期糖基化终产物受体在介导晚期糖基化终产物对糖尿病血管疾病发展的影响中起核心作用。
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本文引用的文献

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Glycaemic control is associated with SARS-CoV-2 breakthrough infections in vaccinated patients with type 2 diabetes.血糖控制与接种疫苗的 2 型糖尿病患者的 SARS-CoV-2 突破感染有关。
Nat Commun. 2022 Apr 28;13(1):2318. doi: 10.1038/s41467-022-30068-2.
2
Impairing RAGE signaling promotes survival and limits disease pathogenesis following SARS-CoV-2 infection in mice.抑制 RAGE 信号通路可促进感染 SARS-CoV-2 的小鼠的存活并限制疾病发病机制。
JCI Insight. 2022 Jan 25;7(2):e155896. doi: 10.1172/jci.insight.155896.
3
Vascular RAGE transports oxytocin into the brain to elicit its maternal bonding behaviour in mice.
血管 RAGE 将催产素转运到大脑中,引发小鼠的母婴结合行为。
Commun Biol. 2019 Feb 25;2:76. doi: 10.1038/s42003-019-0325-6. eCollection 2019.
4
Potential of birds to serve as a pathology-free model of type 2 diabetes, Part 1: Is the apparent absence of the rage gene a factor in the resistance of avian organisms to chronic hyperglycemia?鸟类作为 2 型糖尿病无病理学模型的潜力,第 1 部分:禽类对慢性高血糖的抗性中,rage 基因的明显缺失是否是一个因素?
Rejuvenation Res. 2014 Feb;17(1):54-61. doi: 10.1089/rej.2013.1498.
5
Low-molecular weight fractions of Japanese soy sauce act as a RAGE antagonist via inhibition of RAGE trafficking to lipid rafts.日本酱油的低分子量部分通过抑制 RAGE 向脂筏的转运来充当 RAGE 拮抗剂。
Food Funct. 2013 Dec;4(12):1835-42. doi: 10.1039/c2fo30359k.
6
Septic shock is associated with receptor for advanced glycation end products ligation of LPS.脓毒症休克与晚期糖基化终产物受体对 LPS 的结合有关。
J Immunol. 2011 Mar 1;186(5):3248-57. doi: 10.4049/jimmunol.1002253. Epub 2011 Jan 26.
7
Solution structure of the variable-type domain of the receptor for advanced glycation end products: new insight into AGE-RAGE interaction.晚期糖基化终末产物受体可变型结构域的溶液结构:对AGE-RAGE相互作用的新见解
Biochemistry. 2008 Nov 25;47(47):12299-311. doi: 10.1021/bi800910v.
8
RAGE control of diabetic nephropathy in a mouse model: effects of RAGE gene disruption and administration of low-molecular weight heparin.小鼠模型中晚期糖基化终末产物受体对糖尿病肾病的调控:晚期糖基化终末产物受体基因敲除及低分子量肝素给药的影响
Diabetes. 2006 Sep;55(9):2510-22. doi: 10.2337/db06-0221.
9
Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice.晚期糖基化终末产物受体过表达小鼠中晚期糖尿病肾病的发生与预防
J Clin Invest. 2001 Jul;108(2):261-8. doi: 10.1172/JCI11771.
10
Isolation and characterization of two binding proteins for advanced glycosylation end products from bovine lung which are present on the endothelial cell surface.从牛肺中分离并鉴定出两种存在于内皮细胞表面的晚期糖基化终产物结合蛋白。
J Biol Chem. 1992 Jul 25;267(21):14987-97.