Lau I V, Horsch J D, Viano D C, Andrzejak D V
J Trauma. 1987 Mar;27(3):225-35. doi: 10.1097/00005373-198703000-00001.
Abdominal injury induced by steering wheel contact at a velocity of 32 km/hr was investigated using anesthetized swine as the surrogate on a Hyge sled. The lower rim of the wheel was positioned 5 cm below the xyphoid. By varying wheel stiffness, wheel orientation, and column angle, resultant abdominal injury ranged from fatal or critical to minor or none. Wheel stiffness was found to be the primary determinant of abdominal injury severity. The mechanism of abdominal injury was identified to be the rim impacting the abdomen and exceeding a combined velocity and compression sensitive tolerance limit. Abdominal injury occurred within the initial 15 ms of wheel contact before whole body movement of the surrogate of column compression, which were initiated by hub contact with the thorax. The severity of abdominal injury correlated with the peak viscous response which can be represented by the product of the instantaneous velocity of abdominal deformation and abdominal compression. It did not correlate with spinal acceleration.
以32公里/小时的速度方向盘接触导致的腹部损伤,使用麻醉猪作为替代物在Hyge雪橇上进行了研究。方向盘下缘位于剑突下方5厘米处。通过改变方向盘刚度、方向盘方向和柱角,导致的腹部损伤范围从致命或严重到轻微或无损伤。发现方向盘刚度是腹部损伤严重程度的主要决定因素。确定腹部损伤的机制是轮辋撞击腹部并超过速度和压缩综合敏感耐受极限。腹部损伤发生在轮接触的最初15毫秒内,在柱压缩替代物的全身运动之前,全身运动由轮毂与胸部接触引发。腹部损伤的严重程度与峰值粘性反应相关,峰值粘性反应可以用腹部变形的瞬时速度与腹部压缩的乘积来表示。它与脊柱加速度无关。